CDKN2B methylation status and isolated chromosome 7 abnormalities predict responses to treatment with 5-azacytidine (Q80568676)

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1 January 2020

title

CDKN2B methylation status and isolated chromosome 7 abnormalities predict responses to treatment with 5-azacytidine (English)

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ChEBI release 2020-05-28

K Raj

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A John

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A Ho

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C Chronis

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S Khan

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J Samuel

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S Pomplun

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N S B Thomas

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G J Mufti

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publication date

5 July 2007

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volume

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issue

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page(s)

1937-1944

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Refinement of the international prognostic scoring system (IPSS) by including LDH as an additional prognostic variable to improve risk assessment in patients with primary myelodysplastic syndromes (MDS).

1 January 2020

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Methylation of the p15(INK4B) gene in myelodysplastic syndrome: it can be detected early at diagnosis or during disease progression and is highly associated with leukaemic transformation

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Methylation of p15INK4B is common, is associated with deletion of genes on chromosome arm 7q and predicts a poor prognosis in therapy-related myelodysplasia and acute myeloid leukemia.

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5-Azacytidine and 5-aza-2'-deoxycytidine as inhibitors of DNA methylation: mechanistic studies and their implications for cancer therapy

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Cytogenetic responses in high-risk myelodysplastic syndrome following low-dose treatment with the DNA methylation inhibitor 5-aza-2'-deoxycytidine

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Decitabine improves patient outcomes in myelodysplastic syndromes: results of a phase III randomized study

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Selective variegated methylation of the p15 CpG island in acute myeloid leukemia

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Molecular strategies for detection and quantitation of clonal cytotoxic T-cell responses in aplastic anemia and myelodysplastic syndrome

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DNA methylation changes after 5-aza-2'-deoxycytidine therapy in patients with leukemia

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Demethylation of a hypermethylated P15/INK4B gene in patients with myelodysplastic syndrome by 5-Aza-2'-deoxycytidine (decitabine) treatment

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7 January 2021

Combined DNA methyltransferase and histone deacetylase inhibition in the treatment of myeloid neoplasms

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Functional diversity of DNA methyltransferase inhibitors in human cancer cell lines.

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