Delay of neutrophil apoptosis by the neuropeptide substance P: involvement of caspase cascade (Q73780821)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:11311737%20AND%20SRC:MED&resulttype=core&format=json

6 November 2019

title

Delay of neutrophil apoptosis by the neuropeptide substance P: involvement of caspase cascade (English)

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6 November 2019

S Böckmann

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6 November 2019

J Seep

series ordinal

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6 November 2019

L Jonas

series ordinal

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6 November 2019

publication date

1 April 2001

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6 November 2019

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6 November 2019

volume

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6 November 2019

issue

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6 November 2019

page(s)

661-670

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Neurogenic inflammation and asthma

6 November 2019

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Regulation of granulocyte apoptosis by PKC inhibition and elevation of [Ca2+]i

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Mechanisms Involved in Activation of Human Eosinophil Exocytosis by Substance P: An in Vitro Model of Sensory Neuroimmunomodulation

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Caspases cleave focal adhesion kinase during apoptosis to generate a FRNK-like polypeptide

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Colonic substance P levels are increased in ulcerative colitis and decreased in chronic severe constipation

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Effects of colony-stimulating factors (CSFs) on neutrophil apoptosis: possible roles at inflammation site

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Trophic effect of various neuropeptides on the cultured ventral spinal cord of rat embryo

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Interleukin-8 delays spontaneous and tumor necrosis factor-alpha-mediated apoptosis of human neutrophils.

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Caspase-mediated Cleavage of Focal Adhesion Kinase pp125FAK and Disassembly of Focal Adhesions in Human Endothelial Cell Apoptosis

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Activation and priming of the human neutrophil oxidase response by substance P: distinct signal transduction pathways

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The mammalian tachykinin receptors

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Eosinophils within the healthy or inflamed human intestine produce substance P and vasoactive intestinal peptide

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Membrane-bound peptidases: endocrine, paracrine, and autocrine effects

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Caspase‐mediated proteolysis during apoptosis: insights from apoptotic neutrophils

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Expression of genes involved in initiation, regulation, and execution of apoptosis in human neutrophils and during neutrophil differentiation of HL-60 cells

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Substance P primes the formation of hydrogen peroxide and nitric oxide in human neutrophils.

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NF-kappaB antiapoptosis: induction of TRAF1 and TRAF2 and c-IAP1 and c-IAP2 to suppress caspase-8 activation

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7 January 2021

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7 January 2021

Caspase-dependent cleavage of signaling proteins during apoptosis. A turn-off mechanism for anti-apoptotic signals

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7 January 2021

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Identifiers

DOI

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6 November 2019

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