DNA damage, repair, and antioxidant systems in brain regions: a correlative study (Q73644418)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10754274%20AND%20SRC:MED&resulttype=core&format=json

5 November 2019

title

DNA damage, repair, and antioxidant systems in brain regions: a correlative study (English)

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5 November 2019

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5 November 2019

F Cardozo-Pelaez

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5 November 2019

P J Brooks

series ordinal

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5 November 2019

T Stedeford

series ordinal

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5 November 2019

S Song

series ordinal

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5 November 2019

publication date

1 March 2000

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Free Radical Biology and Medicine

5 November 2019

published in

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5 November 2019

volume

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5 November 2019

issue

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5 November 2019

page(s)

779-785

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Reactive oxygen species and the central nervous system

5 November 2019

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Increased DNA oxidation and decreased levels of repair products in Alzheimer's disease ventricular CSF.

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Cloning and characterization of hOGG1, a human homolog of the OGG1 gene of Saccharomyces cerevisiae

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Changes in the levels of enzymes which modulate the antioxidant balance occur during aging and correlate with cellular damage

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7 January 2021

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Attenuation of age-dependent oxidative damage to DNA and protein in brainstem of Tg Cu/Zn SOD mice

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7 January 2021

In vivo oxidative DNA damage: measurement of 8-hydroxy-2'-deoxyguanosine in DNA and urine by high-performance liquid chromatography with electrochemical detection

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Inhibition of nitrite formation from hydroxylammoniumchloride: a simple assay for superoxide dismutase

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Protein oxidation and aging

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7 January 2021

Free radical oxidation of brain proteins in accelerated senescence and its modulation by N-tert-butyl-alpha-phenylnitrone

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7 January 2021

Effect of age and caloric intake on protein oxidation in different brain regions and on behavioral functions of the mouse

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7 January 2021

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7 January 2021

Relationship between aging and susceptibility to protein oxidative damage.

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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Identifiers

DOI

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5 November 2019

1 reference

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