Functional characterization of p53 molecules expressed in human squamous cell carcinomas of the head and neck (Q42814267)

7 November 2017

title

Functional characterization of p53 molecules expressed in human squamous cell carcinomas of the head and neck (English)

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7 November 2017

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Yeudall WA

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7 November 2017

Jakus J

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7 November 2017

Ensley JF

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7 November 2017

Robbins KC

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7 November 2017

language of work or name

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1 February 1997

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7 November 2017

published in

Molecular Carcinogenesis

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7 November 2017

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7 November 2017

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7 November 2017

page(s)

89-96

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describes a project that uses

7 November 2017

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The tumor suppressor genes

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Thymocyte apoptosis induced by p53-dependent and independent pathways.

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p53 is required for radiation-induced apoptosis in mouse thymocytes

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Wild-type p53 is a cell cycle checkpoint determinant following irradiation

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The p53 proto-oncogene can act as a suppressor of transformation

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Mice deficient for p53 are developmentally normal but susceptible to spontaneous tumours

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Definition of a consensus binding site for p53

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p53 domains: structure, oligomerization, and transformation

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Transcriptional activation by wild-type but not transforming mutants of the p53 anti-oncogene

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Presence of human papillomavirus sequences in tumour-derived human oral keratinocytes expressing mutant p53

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Tyrosine phosphorylation as a marker for aberrantly regulated growth-promoting pathways in cell lines derived from head and neck malignancies

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MTS1/CDK4I is altered in cell lines derived from primary and metastatic oral squamous cell carcinoma

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Oncogenic forms of p53 inhibit p53-regulated gene expression

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An infrequent point mutation of the p53 gene in human nasopharyngeal carcinoma

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Functional characterization in vivo of mutant p53 molecules derived from squamous cell carcinomas of the head and neck

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Progression toward tumor cell phenotype is enhanced by overexpression of a mutant p53 tumor-suppressor gene isolated from nasopharyngeal carcinoma

21 January 2018

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The p53 status of cultured human premalignant oral keratinocytes

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Cellular immortality: a late event in the progression of human squamous cell carcinoma of the head and neck associated with p53 alteration and a high frequency of allele loss.

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Inactivation of the p53 gene by either mutation or HPV infection is extremely frequent in human oral squamous cell carcinoma cell lines.

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10.1002/(SICI)1098-2744(199702)18:2<89::AID-MC4>3.0.CO;2-L

21 January 2018

Identifiers

DOI

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7 November 2017

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