Neuronal protein tyrosine phosphatase 1B deficiency results in inhibition of hypothalamic AMPK and isoform-specific activation of AMPK in peripheral tissues (Q41818058)

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scientific article published on 15 June 2009

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English	Neuronal protein tyrosine phosphatase 1B deficiency results in inhibition of hypothalamic AMPK and isoform-specific activation of AMPK in peripheral tissues	scientific article published on 15 June 2009

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31 January 2020

Neuronal protein tyrosine phosphatase 1B deficiency results in inhibition of hypothalamic AMPK and isoform-specific activation of AMPK in peripheral tissues (English)

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Thomas Pulinilkunnil

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Mirela Delibegovic

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Bingzhong Xue

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Incoronata Murano

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Huamei He

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Yasuhiko Minokoshi

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Kenji Asakura

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Anna Lee

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Fawaz Haj

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Noboru Furukawa

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Karyn J Catalano

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James A Balschi

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Benjamin G Neel

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Molecular and Cellular Biology

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31 January 2020

Liver-specific deletion of protein-tyrosine phosphatase 1B (PTP1B) improves metabolic syndrome and attenuates diet-induced endoplasmic reticulum stress

1 reference

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6 June 2018

AMP-activated protein kinase in skeletal muscle: from structure and localization to its role as a master regulator of cellular metabolism

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Downregulation of AMP-activated protein kinase by Cidea-mediated ubiquitination and degradation in brown adipose tissue

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6 June 2018

Improved glucose homeostasis in mice with muscle-specific deletion of protein-tyrosine phosphatase 1B.

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AMPK is essential for energy homeostasis regulation and glucose sensing by POMC and AgRP neurons

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AMP-activated protein kinase (AMPK) action in skeletal muscle via direct phosphorylation of PGC-1alpha

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Investigating the mechanism for AMP activation of the AMP-activated protein kinase cascade

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Role of AMPKalpha2 in basal, training-, and AICAR-induced GLUT4, hexokinase II, and mitochondrial protein expression in mouse muscle

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AMPK integrates nutrient and hormonal signals to regulate food intake and energy balance through effects in the hypothalamus and peripheral tissues

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AMPK activation increases fatty acid oxidation in skeletal muscle by activating PPARalpha and PGC-1.

1 reference

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Deficiency of LKB1 in heart prevents ischemia-mediated activation of AMPKalpha2 but not AMPKalpha1.

1 reference

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Involvement of the small protein tyrosine phosphatases TC-PTP and PTP1B in signal transduction and diseases: from diabetes, obesity to cell cycle, and cancer

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Endurance training increases skeletal muscle LKB1 and PGC-1alpha protein abundance: effects of time and intensity

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Deficiency of LKB1 in skeletal muscle prevents AMPK activation and glucose uptake during contraction

1 reference

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Mechanism of protein tyrosine phosphatase 1B-mediated inhibition of leptin signalling

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Stearoyl-CoA desaturase 1 deficiency increases fatty acid oxidation by activating AMP-activated protein kinase in liver.

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AMP-kinase regulates food intake by responding to hormonal and nutrient signals in the hypothalamus

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The tumor suppressor LKB1 kinase directly activates AMP-activated kinase and regulates apoptosis in response to energy stress

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LKB1 is the upstream kinase in the AMP-activated protein kinase cascade

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Complexes between the LKB1 tumor suppressor, STRAD alpha/beta and MO25 alpha/beta are upstream kinases in the AMP-activated protein kinase cascade

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PTP1B: from the sidelines to the front lines!

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Antisense protein tyrosine phosphatase 1B reverses activation of p38 mitogen-activated protein kinase in liver of ob/ob mice

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6 June 2018

Reduction of protein tyrosine phosphatase 1B increases insulin-dependent signaling in ob/ob mice

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6 June 2018

Protein-tyrosine phosphatase 1B (PTP1B): a novel therapeutic target for type 2 diabetes mellitus, obesity and related states of insulin resistance

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6 June 2018

AMP kinase is required for mitochondrial biogenesis in skeletal muscle in response to chronic energy deprivation

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6 June 2018

Protein tyrosine phosphatase 1B negatively regulates leptin signaling in a hypothalamic cell line

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Attenuation of leptin action and regulation of obesity by protein tyrosine phosphatase 1B.

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PTP1B regulates leptin signal transduction in vivo

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Chronic activation of AMP kinase results in NRF-1 activation and mitochondrial biogenesis

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TYK2 and JAK2 are substrates of protein-tyrosine phosphatase 1B

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Structural and evolutionary relationships among protein tyrosine phosphatase domains

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Neural influences on trophic changes in brown adipose tissue during cold acclimation

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The bioenergetics of brown fat mitochondria from UCP1-ablated mice. Ucp1 is not involved in fatty acid-induced de-energization ("uncoupling")

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Myocardial ischemia differentially regulates LKB1 and an alternate 5'-AMP-activated protein kinase kinase

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Identifiers

10.1128/MCB.01914-08

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Europe PubMed Central

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31 January 2020

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Europe PubMed Central

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31 January 2020

PubMed publication ID

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:19528236%20AND%20SRC:MED&resulttype=core&format=json

31 January 2020

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