Upregulation of enzymatic activity by interleukin-1 in osteoarthritis (Q41479491)

1 October 2017

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title

Upregulation of enzymatic activity by interleukin-1 in osteoarthritis (English)

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1 October 2017

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Chevalier X

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1 October 2017

publication date

1 January 1997

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Biomedicine and Pharmacotherapy

1 October 2017

published in

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1 October 2017

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1 October 2017

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1 October 2017

page(s)

58-62

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Structure and biology of cartilage and bone matrix noncollagenous macromolecules

1 October 2017

cites work

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Stimulation of the secretion of latent cysteine proteinase activity by tumor necrosis factor alpha and interleukin-1

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Imbalance between the mechanisms of activation and inhibition of metalloproteases in the early lesions of experimental osteoarthritis

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The superficial layer of human articular cartilage is more susceptible to interleukin-1-induced damage than the deeper layers

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Interleukin-1 beta-modulated gene expression in immortalized human chondrocytes

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Interleukin-1 stimulates and all-trans-retinoic acid inhibits collagenase gene expression through its 5' activator protein-1-binding site

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Inhibition of cartilage proteoglycan release by a specific inactivator of cathepsin B and an inhibitor of matrix metalloproteinases. Evidence for two converging pathways of chondrocyte-mediated proteoglycan degradation

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Modulation of the catabolic effects of interleukin-1 beta on human articular chondrocytes by transforming growth factor-beta

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Direct evidence for active metalloproteinases mediating matrix degradation in interleukin 1-stimulated human articular cartilage

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Interleukin-1-Induced Prostaglandin E2 Biosynthesis in Human Synovial Cells Involves the Activation of Cytosolic Phospholipase A2 and Cyclooxygenase-2

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Some recombinant human cytokines stimulate glycosaminoglycan synthesis in human synovial fibroblast cultures and inhibit it in human articular cartilage cultures

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The effect of interleukin‐1 on cytokine gene expression in cultured human articular chondrocytes analyzed by messenger rna phenotyping

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Interactions between interleukin-1 and basic fibroblast growth factor on articular chondrocytes. Effects on cell growth, prostanoid production, and receptor modulation

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TGF-beta 1 inhibition of transin/stromelysin gene expression is mediated through a Fos binding sequence

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7 January 2021

Expression of IL-1 genes in human and bovine chondrocytes: a mechanism for autocrine control of cartilage matrix degradation

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In vivo studies of cartilage regeneration after damage induced by catabolin/interleukin-1.

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Identifiers

DOI

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1 October 2017

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