Zinc status affects p53, gadd45, and c-fos expression and caspase-3 activity in human bronchial epithelial cells (Q40786496)

23 September 2017

title

Zinc status affects p53, gadd45, and c-fos expression and caspase-3 activity in human bronchial epithelial cells (English)

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23 September 2017

Fanzo JC

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23 September 2017

Reaves SK

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23 September 2017

Cui L

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23 September 2017

Zhu L

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23 September 2017

Wu JY

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23 September 2017

Wang YR

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23 September 2017

Lei KY

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23 September 2017

publication date

1 September 2001

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American Journal of Physiology - Cell Physiology

23 September 2017

published in

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23 September 2017

volume

281

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23 September 2017

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23 September 2017

page(s)

C751-7

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Zinc ions prevent processing of caspase-3 during apoptosis induced by geranylgeraniol in HL-60 cells

23 September 2017

cites work

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Expression of human p53 requires synergistic activation of transcription from the p53 promoter by AP-1, NF-kappaB and Myc/Max

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Relationship between p53 mutation incidence in oral cavity squamous cell carcinomas and patient tobacco use.

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Tumor suppressor p53 is a direct transcriptional activator of the human bax gene

21 January 2018

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Zinc is a potent inhibitor of the apoptotic protease, caspase-3. A novel target for zinc in the inhibition of apoptosis

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Zinc deficiency affects cell cycle and deoxythymidine kinase gene expression in HUT-78 cells

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Interaction of the p53-regulated protein Gadd45 with proliferating cell nuclear antigen

21 January 2018

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p53: a frequent target for genetic abnormalities in lung cancer.

21 January 2018

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Modulation of p53 protein conformation and DNA-binding activity by intracellular chelation of zinc.

21 January 2018

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Deletion of a DNA sequence at the chromosomal region 3p21 in all major types of lung cancer

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10.1152/AJPCELL.2001.281.3.C751

21 January 2018

Identifiers

DOI

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