Ability of GDNF to diminish free radical production leads to protection against kainate-induced excitotoxicity in hippocampus (Q40571121)

21 September 2017

title

Ability of GDNF to diminish free radical production leads to protection against kainate-induced excitotoxicity in hippocampus (English)

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21 September 2017

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Henrich Cheng

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21 September 2017

Yu-Show Fu

series ordinal

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21 September 2017

Jiun-Wen Guo

series ordinal

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21 September 2017

publication date

1 January 2004

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21 September 2017

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21 September 2017

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21 September 2017

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21 September 2017

page(s)

77-86

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Estimation of nuclear population from microtome sections

21 September 2017

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Calcium-dependent free radical generation in cultured retinal neurons injured by kainate

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Oxidative stress is involved in seizure-induced neurodegeneration in the kindling model of epilepsy

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Inhibitors of NO-synthase and donors of NO modulate kainic acid-induced damage in the rat hippocampus

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The relationship between stress protein induction and the oxidative defense system in the rat hippocampus following kainic acid administration

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Glutamate regulation of GDNF gene expression in the striatum and primary striatal astrocytes.

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Neurons of the hippocampal formation express glial cell line-derived neurotrophic factor messenger RNA in response to kainate-induced excitation

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Chronic exposure of kainate and NBQX changes AMPA toxicity in hippocampal slice cultures

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Nitric oxide synthase inhibitors L-NAME and 7-nitroindazole protect rat hippocampus against kainate-induced excitotoxicity

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Kainic acid may enhance hippocampal NO generation of awake rats in a seizure stage-related fashion

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Kainate-evoked secondary gene expression in the rat hippocampus

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Time course of transient expression of GDNF protein in rat granule cells of the bilateral dentate gyri after unilateral intrahippocampal kainic acid injection

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Kainic acid-induced lipid peroxidation: protection with butylated hydroxytoluene and U78517F in primary cultures of cerebellar granule cells

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Formation of free hydroxyl radicals after pentylenetetrazol-induced seizure and kindling

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Development of high affinity kainate binding sites in human and rat hippocampi.

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Generation of lipid radicals in the hippocampal extracellular space during kainic acid-induced seizures in rats

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NMDA and kainate induce internucleosomal DNA cleavage associated with both apoptotic and necrotic cell death in the neonatal rat brain

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Identifiers

DOI

10.1002/HIPO.10145

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21 September 2017

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