Over-expression of miR-196b-5p is significantly associated with the progression of myelodysplastic syndrome (Q48693128)

14 February 2018

title

Over-expression of miR-196b-5p is significantly associated with the progression of myelodysplastic syndrome (English)

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14 February 2018

author name string

Jing Wen

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14 February 2018

Ying Huang

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14 February 2018

Hongying Li

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14 February 2018

Xupai Zhang

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14 February 2018

Peng Cheng

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14 February 2018

Donghong Deng

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14 February 2018

Zhigang Peng

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14 February 2018

Jun Luo

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14 February 2018

Weihua Zhao

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14 February 2018

Yongrong Lai

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14 February 2018

Zhenfang Liu

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14 February 2018

publication date

21 February 2017

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14 February 2018

International Journal of Hematology

published in

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14 February 2018

volume

105

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14 February 2018

issue

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14 February 2018

page(s)

777-783

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14 February 2018

Pathogenesis of myelodysplastic syndromes: an overview of molecular and non-molecular aspects of the disease

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Identification of a risk dependent microRNA expression signature in myelodysplastic syndromes

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Myeloid cell differentiation arrest by miR-125b-1 in myelodysplastic syndrome and acute myeloid leukemia with the t(2;11)(p21;q23) translocation

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Regulation of mir-196b by MLL and its overexpression by MLL fusions contributes to immortalization

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Circulating microRNAs as stable blood-based markers for cancer detection

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Bone progenitor dysfunction induces myelodysplasia and secondary leukaemia.

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12 December 2020

Aberrant microRNA expression pattern in myelodysplastic bone marrow cells

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12 December 2020

Clonal origin and evolution of myelodysplastic syndrome analyzed by dysplastic morphology and fluorescence in situ hybridization

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12 December 2020

Use of an aqueous soluble tetrazolium/formazan assay to measure viability and proliferation of lymphokine-dependent cell lines

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12 December 2020

Myelodysplastic syndromes

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12 December 2020

In ovo application of antagomiRs indicates a role for miR-196 in patterning the chick axial skeleton through Hox gene regulation

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12 December 2020

10.1007/S12185-017-2201-9

Identifiers

DOI

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14 February 2018

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14 February 2018