Deletion of the c-Jun N-terminal kinase 3 gene protects neonatal mice against cerebral hypoxic-ischaemic injury. (Q48388270)

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21 December 2019

title

Deletion of the c-Jun N-terminal kinase 3 gene protects neonatal mice against cerebral hypoxic-ischaemic injury (English)

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21 December 2019

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21 December 2019

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21 December 2019

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21 December 2019

Katarina G Brywe

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21 December 2019

Grisha Pirianov

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21 December 2019

Richard A Flavell

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21 December 2019

Huseyin Mehmet

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21 December 2019

publication date

25 October 2006

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Journal of Cerebral Blood Flow & Metabolism

21 December 2019

published in

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21 December 2019

volume

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21 December 2019

issue

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21 December 2019

page(s)

1022-1032

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Characterization of the c-Jun N-terminal kinase-BimEL signaling pathway in neuronal apoptosis.

21 December 2019

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Jun NH2-terminal kinase phosphorylation of p53 on Thr-81 is important for p53 stabilization and transcriptional activities in response to stress.

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Control of death receptor and mitochondrial-dependent apoptosis by c-Jun N-terminal kinase in hippocampal CA1 neurones following global transient ischaemia

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Evidence that p38 mitogen-activated protein kinase contributes to neonatal hypoxic-ischemic brain injury

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Role of caspase-3 activation in cerebral ischemia-induced neurodegeneration in adult and neonatal brain

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Dominant-negative c-Jun promotes neuronal survival by reducing BIM expression and inhibiting mitochondrial cytochrome c release

21 January 2018

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Opposing effects of ERK and JNK-p38 MAP kinases on apoptosis

21 January 2018

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21 January 2018

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21 January 2018

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7 January 2021

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