Neuronal nicotinic receptor deficits in Alzheimer patients with the Swedish amyloid precursor protein 670/671 mutation (Q48275764)

10 February 2018

title

Neuronal nicotinic receptor deficits in Alzheimer patients with the Swedish amyloid precursor protein 670/671 mutation (English)

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10 February 2018

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A Nordberg

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10 February 2018

author name string

A Marutle

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10 February 2018

U Warpman

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10 February 2018

N Bogdanovic

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L Lannfelt

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10 February 2018

language of work or name

English

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publication date

1 March 1999

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10 February 2018

Journal of Neurochemistry

published in

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volume

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issue

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10 February 2018

page(s)

1161-1169

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10 February 2018

Beta-amyloid peptides as direct cholinergic neuromodulators: a missing link?

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A large Swedish family with Alzheimer's disease with a codon 670/671 amyloid precursor protein mutation. A clinical and genealogical investigation.

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Evidence that tobacco smoking increases the density of (-)-[3H]nicotine binding sites in human brain

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Protective effect of apolipoprotein E type 2 allele for late onset Alzheimer disease

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Alzheimer's Disease: A Disorder of Cortical Cholinergic Innervation

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Acetylcholine-activated inward current induces cytosolic Ca2+ mobilization in mouse C2C12 myotubes.

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Amyloid precursor protein mutation causes Alzheimer's disease in a Swedish family

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Nicotinic systems and cognitive function

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Regional distribution of subtypes of nicotinic receptors in human brain and effect of aging studied by (+/-)-[3H]epibatidine

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beta-Amyloid precursor protein metabolites and loss of neuronal Ca2+ homeostasis in Alzheimer's disease.

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Distribution of nicotinic receptors in the human hippocampus and thalamus

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Nicotine inhibits amyloid formation by the beta-peptide.

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Increased amyloid beta-peptide deposition in cerebral cortex as a consequence of apolipoprotein E genotype in late-onset Alzheimer disease

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21 January 2018

Identifiers

DOI

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10 February 2018

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10 February 2018