Bone Resorption Is Regulated by Circadian Clock in Osteoblasts (Q47859307)

6 February 2018

title

Bone Resorption Is Regulated by Circadian Clock in Osteoblasts (English)

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Yukio Yoneda

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author name string

Takeshi Takarada

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Cheng Xu

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Hiroki Ochi

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Ryota Nakazato

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Daisuke Yamada

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Saki Nakamura

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Ayumi Kodama

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Shigeki Shimba

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Kazuya Fukasawa

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Kakeru Ozaki

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Takashi Iezaki

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Koichi Fujikawa

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Rika Numano

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Akiko Hida

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Hajime Tei

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Shu Takeda

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Eiichi Hinoi

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publication date

7 December 2016

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Journal of Bone and Mineral Research

published in

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volume

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issue

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page(s)

872-881

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Genetic regulation of osteoclast development and function

cites work

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Control of osteoblast function and regulation of bone mass

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Genetic control of bone formation

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Disorders of Bone Remodeling

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Clock genes in the heart: characterization and attenuation with hypertrophy

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Extensive and divergent circadian gene expression in liver and heart

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Brain and muscle Arnt-like protein-1 (BMAL1), a component of the molecular clock, regulates adipogenesis

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Resetting central and peripheral circadian oscillators in transgenic rats

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The genetics of mammalian circadian order and disorder: implications for physiology and disease

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Clock genes influence gene expression in growth plate and endochondral ossification in mice

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The molecular clock mediates leptin-regulated bone formation.

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Deficient of a clock gene, brain and muscle Arnt-like protein-1 (BMAL1), induces dyslipidemia and ectopic fat formation

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Intrinsic circadian clock of the mammalian retina: importance for retinal processing of visual information

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Mouse alpha1(I)-collagen promoter is the best known promoter to drive efficient Cre recombinase expression in osteoblast

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Reduced chondrocyte proliferation and chondrodysplasia in mice lacking the integrin-linked kinase in chondrocytes

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DMP1-targeted Cre expression in odontoblasts and osteocytes

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Osteoblastic γ-aminobutyric acid, type B receptors negatively regulate osteoblastogenesis toward disturbance of osteoclastogenesis mediated by receptor activator of nuclear factor κB ligand in mouse bone

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The Transcriptional Modulator Interferon-Related Developmental Regulator 1 in Osteoblasts Suppresses Bone Formation and Promotes Bone Resorption

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The natural polyamines spermidine and spermine prevent bone loss through preferential disruption of osteoclastic activation in ovariectomized mice.

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Standardized nomenclature, symbols, and units for bone histomorphometry: a 2012 update of the report of the ASBMR Histomorphometry Nomenclature Committee

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Guidelines for assessment of bone microstructure in rodents using micro-computed tomography

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Up-regulation of per mRNA expression by parathyroid hormone through a protein kinase A-CREB-dependent mechanism in chondrocytes.

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Evidence for osteocyte regulation of bone homeostasis through RANKL expression

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Matrix-embedded cells control osteoclast formation

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Mice lacking the vitamin D receptor exhibit impaired bone formation, uterine hypoplasia and growth retardation after weaning

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Vitamin D(3) augments osteoclastogenesis via vitamin D-responsive element of mouse RANKL gene promoter

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Osteoblast precursors, but not mature osteoblasts, move into developing and fractured bones along with invading blood vessels.

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Osx-Cre targets multiple cell types besides osteoblast lineage in postnatal mice

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Genetic mouse models for bone studies--strengths and limitations

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Circadian Clock Regulates Bone Resorption in Mice

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21 January 2018

Identifiers

DOI

10.1002/JBMR.3053

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