Roles of Bcl-2 and caspases in hypoxia-induced neuronal cell death: a possible neuroprotective mechanism of peptide growth factors (Q47760430)

5 February 2018

title

Roles of Bcl-2 and caspases in hypoxia-induced neuronal cell death: a possible neuroprotective mechanism of peptide growth factors (English)

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5 February 2018

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Tamatani M

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5 February 2018

Ogawa S

series ordinal

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5 February 2018

Tohyama M

series ordinal

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5 February 2018

publication date

1 July 1998

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5 February 2018

published in

Molecular Brain Research

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5 February 2018

volume

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5 February 2018

issue

1-2

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5 February 2018

page(s)

27-39

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5 February 2018

Activation of the CED3/ICE-related protease CPP32 in cerebellar granule neurons undergoing apoptosis but not necrosis

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Mechanisms of delayed cell death following hypoxic-ischemic injury in the immature rat: evidence for apoptosis during selective neuronal loss

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Expression of the apoptosis-effector gene, Bax, is up-regulated in vulnerable hippocampal CA1 neurons following global ischemia

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Expression of a dominant negative mutant of interleukin-1 beta converting enzyme in transgenic mice prevents neuronal cell death induced by trophic factor withdrawal and ischemic brain injury

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Peptide growth factors protect against ischemia in culture by preventing nitric oxide toxicity

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7 January 2021

ICE-like proteases execute the neuronal death program.

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7 January 2021

Inhibition of Ced-3/ICE-related proteases does not prevent cell death induced by oncogenes, DNA damage, or the Bcl-2 homologue Bak

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7 January 2021

Peptide inhibitors of the ice protease family arrest programmed cell death of motoneurons in vivo and in vitro

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7 January 2021

Hypoxia modulates the barrier and coagulant function of cultured bovine endothelium. Increased monolayer permeability and induction of procoagulant properties

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7 January 2021

Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

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7 January 2021

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A novel heterodimeric cysteine protease is required for interleukin-1 beta processing in monocytes

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7 January 2021

The t(14;18) chromosome translocations involved in B-cell neoplasms result from mistakes in VDJ joining

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7 January 2021

Ich-1, an Ice/ced-3-related gene, encodes both positive and negative regulators of programmed cell death

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7 January 2021

Cell death: the significance of apoptosis

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7 January 2021

Basic fibroblast growth factor prevents thalamic degeneration after cortical infarction

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7 January 2021

The C. elegans cell death gene ced-3 encodes a protein similar to mammalian interleukin-1 beta-converting enzyme

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7 January 2021

10.1016/S0169-328X(98)00095-3

Identifiers

DOI

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5 February 2018

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5 February 2018