The Brucella effector protein TcpB induces degradation of inflammatory caspases and thereby subverts non-canonical inflammasome activation in macrophages. (Q46276836)

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scientific article published on 23 October 2017

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English	The Brucella effector protein TcpB induces degradation of inflammatory caspases and thereby subverts non-canonical inflammasome activation in macrophages.	scientific article published on 23 October 2017

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8 April 2020

The Brucella effector protein TcpB induces degradation of inflammatory caspases and thereby subverts non-canonical inflammasome activation in macrophages (English)

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8 April 2020

inflammasome complex

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Girish Radhakrishnan

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8 April 2020

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Padmaja Jakka

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8 April 2020

Swapna Namani

2

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8 April 2020

Subathra Murugan

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8 April 2020

Nivedita Rai

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8 April 2020

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23 October 2017

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8 April 2020

Journal of Biological Chemistry

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8 April 2020

292

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8 April 2020

50

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8 April 2020

20613-20627

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8 April 2020

Pyroptosis: Gasdermin-Mediated Programmed Necrotic Cell Death

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Bacterial virulence factor inhibits caspase-4/11 activation in intestinal epithelial cells

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Inflammasome-activated gasdermin D causes pyroptosis by forming membrane pores

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Converging roles of caspases in inflammasome activation, cell death and innate immunity

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Human caspase-4 and caspase-5 regulate the one-step non-canonical inflammasome activation in monocytes

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Cleavage of GSDMD by inflammatory caspases determines pyroptotic cell death

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NLRP3 inflammasome activation downstream of cytoplasmic LPS recognition by both caspase-4 and caspase-5.

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Revisiting caspases in sepsis

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Noncanonical inflammasome activation of caspase-4/caspase-11 mediates epithelial defenses against enteric bacterial pathogens

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Brucella induces an unfolded protein response via TcpB that supports intracellular replication in macrophages

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Noncanonical inflammasome activation by intracellular LPS independent of TLR4.

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26 August 2018

BtpB, a novel Brucella TIR-containing effector protein with immune modulatory functions

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Critical role of ASC inflammasomes and bacterial type IV secretion system in caspase-1 activation and host innate resistance to Brucella abortus infection

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