Sustained hepatic expression of FoxM1B in transgenic mice has minimal effects on hepatocellular carcinoma development but increases cell proliferation rates in preneoplastic and early neoplastic lesions (Q44588837)

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scientific article published in September 2003

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English	Sustained hepatic expression of FoxM1B in transgenic mice has minimal effects on hepatocellular carcinoma development but increases cell proliferation rates in preneoplastic and early neoplastic lesions	scientific article published in September 2003

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

4 December 2017

Sustained hepatic expression of FoxM1B in transgenic mice has minimal effects on hepatocellular carcinoma development but increases cell proliferation rates in preneoplastic and early neoplastic lesions (English)

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Europe PubMed Central

PubMed publication ID

4 December 2017

hepatocellular carcinoma

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author name string

Olga A Kalinina

1

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4 December 2017

Sergey A Kalinin

2

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4 December 2017

Evelyne W Polack

3

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Europe PubMed Central

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4 December 2017

Igor Mikaelian

4

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4 December 2017

Suchismita Panda

5

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4 December 2017

Robert H Costa

6

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4 December 2017

Guy R Adami

7

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4 December 2017

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publication date

1 September 2003

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4 December 2017

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4 December 2017

22

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40

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6266-6276

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4 December 2017

Epidemiology of primary liver cancer

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Coexpression of C- myc and transforming growth factor alfa in the liver promotes early replicative senescence and diminishes regenerative capacity after partial hepatectomy in transgenic mice

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Liver regeneration

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Delayed and reduced cell replication and diminishing levels of DNA polymerase-alpha in regenerating liver of aging mice

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Inhibition of WY-14,643 induced hepatic lesion growth in mice by rotenone

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The Winged-Helix Transcription Factor Trident is Expressed in Actively Dividing Lymphocytes

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7 January 2021

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Over-expression of FoxM1 stimulates cyclin B1 expression

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Mitotic misregulation and human aging

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Bcl-2 expression inhibits liver carcinogenesis and delays the development of proliferating foci

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Pathways of progression in hepatocarcinogenesis

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Stage-specific gene expression during hepatocarcinogenesis in the rat.

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Agents that cause DNA double strand breaks lead to p16INK4a enrichment and the premature senescence of normal fibroblasts

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Rapid development of hepatic tumors in transforming growth factor alpha transgenic mice associated with increased cell proliferation in precancerous hepatocellular lesions initiated by N-nitrosodiethylamine and promoted by phenobarbital

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Earlier expression of the transcription factor HFH-11B diminishes induction of p21(CIP1/WAF1) levels and accelerates mouse hepatocyte entry into S-phase following carbon tetrachloride liver injury

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The Forkhead Box m1b transcription factor is essential for hepatocyte DNA replication and mitosis during mouse liver regeneration

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Increased hepatic Forkhead Box M1B (FoxM1B) levels in old-aged mice stimulated liver regeneration through diminished p27Kip1 protein levels and increased Cdc25B expression

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Increased levels of forkhead box M1B transcription factor in transgenic mouse hepatocytes prevent age-related proliferation defects in regenerating liver

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10.1038/SJ.ONC.1206640

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Europe PubMed Central

PubMed publication ID

4 December 2017

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PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

4 December 2017

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