Effects of 17 alpha-methyltestosterone on uterine morphology and heat shock protein expression are mediated through estrogen and androgen receptors (Q44317855)

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scientific article published in November 2002

Language	Label	Description	Also known as
English	Effects of 17 alpha-methyltestosterone on uterine morphology and heat shock protein expression are mediated through estrogen and androgen receptors	scientific article published in November 2002

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scholarly article

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Europe PubMed Central

PubMed publication ID

1 December 2017

Effects of 17 alpha-methyltestosterone on uterine morphology and heat shock protein expression are mediated through estrogen and androgen receptors (English)

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Europe PubMed Central

PubMed publication ID

1 December 2017

author name string

Andriana D Papaconstantinou

1

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Europe PubMed Central

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1 December 2017

Thomas H Umbreit

2

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Europe PubMed Central

PubMed publication ID

1 December 2017

Peter L Goering

3

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Europe PubMed Central

PubMed publication ID

1 December 2017

Ken M Brown

4

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Europe PubMed Central

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1 December 2017

publication date

1 November 2002

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Europe PubMed Central

PubMed publication ID

1 December 2017

The Journal of Steroid Biochemistry and Molecular Biology

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1 December 2017

82

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1 December 2017

4-5

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Europe PubMed Central

PubMed publication ID

1 December 2017

305-314

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PubMed publication ID

1 December 2017

Conservative treatment of endometriosis externa: the effects of methyltestosterone therapy

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Binding of testosterone to cytoplasmic components of the immature rat uterus

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A repeated 28-day oral dose toxicity study of 17alpha-methyltestosterone in rats, based on the 'enhanced OECD Test Guideline 407' for screening the endocrine-disrupting chemicals

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17alpha-methyl testosterone is a competitive inhibitor of aromatase activity in Jar choriocarcinoma cells and macrophage-like THP-1 cells in culture

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Effects of beta-estradiol and bisphenol A on heat shock protein levels and localization in the mouse uterus are antagonized by the antiestrogen ICI 182,780.

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The effects of 17 alpha-methyltestosterone, methandrostenolone, and nandrolone decanoate on the rat estrous cycle.

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Aromatase inhibitor and 17alpha-methyltestosterone cause sex-reversal from genetical females to phenotypic males and suppression of P450 aromatase gene expression in Japanese flounder (Paralichthys olivaceus)

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The glucocorticoid antagonist 17 alpha-methyltestosterone binds to the 10 S glucocorticoid receptor and blocks agonist-mediated dissociation of the 10 S oligomer to the 4 S deoxyribonucleic acid-binding subunit

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Evaluation of a Tier I screening battery for detecting endocrine-active compounds (EACs) using the positive controls testosterone, coumestrol, progesterone, and RU486

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Identifiers

10.1016/S0960-0760(02)00221-2

1 reference

Europe PubMed Central

PubMed publication ID

1 December 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

1 December 2017

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