Triplication of a 21q22 region contributes to B cell transformation through HMGN1 overexpression and loss of histone H3 Lys27 trimethylation (Q33691942)

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English	Triplication of a 21q22 region contributes to B cell transformation through HMGN1 overexpression and loss of histone H3 Lys27 trimethylation	scientific article

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28 July 2017

Triplication of a 21q22 region contributes to B cell transformation through HMGN1 overexpression and loss of histone H3 Lys27 trimethylation (English)

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Kimberly Stegmaier

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Kimberly Stegmaier

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Gabriela Alexe

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Sébastien Malinge

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Sébastien Malinge

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John D Crispino

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John D Crispino

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Geertruy Te Kronnie

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Geertruy te Kronnie

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William C Hahn

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Anna C Schinzel

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Anna C Schinzel

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David M Weinstock

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David M Weinstock

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Donna Neuberg

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Andrew A Lane

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Bjoern Chapuy

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Charles Y Lin

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Trevor Tivey

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Hubo Li

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Elizabeth C Townsend

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Diederik van Bodegom

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Tovah A Day

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Shuo-Chieh Wu

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Huiyun Liu

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Akinori Yoda

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Timothy J Sullivan

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Jordan E Taylor

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Jacob D Jaffe

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Michael Bustin

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Shai Izraeli

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Marian H Harris

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Kristen E Stevenson

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Lewis B Silverman

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Stephen E Sallan

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James E Bradner

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David Pellman

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20 April 2014

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Nature Genetics

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46

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6

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618-623

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28 July 2017

Selective inhibition of tumor oncogenes by disruption of super-enhancers

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Quantitative assessment of chromatin immunoprecipitation grade antibodies directed against histone modifications reveals patterns of co-occurring marks on histone protein molecules

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Effects of HMGN variants on the cellular transcription profile

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Down syndrome acute lymphoblastic leukemia, a highly heterogeneous disease in which aberrant expression of CRLF2 is associated with mutated JAK2: a report from the International BFM Study Group

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Mutational spectrum at GATA1 provides insights into mutagenesis and leukemogenesis in Down syndrome

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Pre-B cell receptor-mediated cell cycle arrest in Philadelphia chromosome-positive acute lymphoblastic leukemia requires IKAROS function

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Functional expression and germline transmission of a human chromosome fragment in chimaeric mice

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28 July 2017

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