Glial cell lineage expression of mutant ataxin-1 and huntingtin induces developmental and late-onset neuronal pathologies in Drosophila models (Q33402470)

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English	Glial cell lineage expression of mutant ataxin-1 and huntingtin induces developmental and late-onset neuronal pathologies in Drosophila models	scientific article

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scholarly article

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Europe PubMed Central

PubMed publication ID

26 July 2017

Glial cell lineage expression of mutant ataxin-1 and huntingtin induces developmental and late-onset neuronal pathologies in Drosophila models (English)

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Europe PubMed Central

PubMed publication ID

26 July 2017

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Erich E Wanker

4

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Takuya Tamura

1

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Europe PubMed Central

PubMed publication ID

26 July 2017

Masaki Sone

2

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Europe PubMed Central

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26 July 2017

Mayumi Yamashita

3

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26 July 2017

Hitoshi Okazawa

5

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26 July 2017

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23 January 2009

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Europe PubMed Central

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26 July 2017

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Europe PubMed Central

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26 July 2017

4

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Europe PubMed Central

PubMed publication ID

26 July 2017

1

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Europe PubMed Central

PubMed publication ID

26 July 2017

e4262

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Europe PubMed Central

PubMed publication ID

26 July 2017

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Creative Commons Attribution 4.0 International

23 January 2009

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April 2022 Public Data File from Crossref

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Opposing effects of polyglutamine expansion on native protein complexes contribute to SCA1

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glial cells missing: a binary switch between neuronal and glial determination in Drosophila.

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Novel gcm-dependent lineages in the postembryonic nervous system of Drosophila melanogaster

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AKT-sensitive or insensitive pathways of toxicity in glial cells and neurons in Drosophila models of Huntington's disease.

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Glial and neuronal expression of polyglutamine proteins induce behavioral changes and aggregate formation in Drosophila.

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ATAXIN-1 interacts with the repressor Capicua in its native complex to cause SCA1 neuropathology

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Mutant huntingtin promotes the fibrillogenesis of wild-type huntingtin: a potential mechanism for loss of huntingtin function in Huntington's disease

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Geldanamycin activates a heat shock response and inhibits huntingtin aggregation in a cell culture model of Huntington's disease

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Identification of genes that modify ataxin-1-induced neurodegeneration.

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Gliogenesis depends on glide/gcm through asymmetric division of neuroglioblasts.

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Tramtrack controls glial number and identity in the Drosophila embryonic CNS.

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31 October 2018

Identifiers

10.1371/JOURNAL.PONE.0004262

1 reference

Europe PubMed Central

PubMed publication ID

26 July 2017

2009PLoSO...4.4262T

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PMC publication ID

1 reference

Europe PubMed Central

PubMed publication ID

26 July 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

26 July 2017

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