Incompetence of neutrophils to invasive group A streptococcus is attributed to induction of plural virulence factors by dysfunction of a regulator (Q33378516)

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English	Incompetence of neutrophils to invasive group A streptococcus is attributed to induction of plural virulence factors by dysfunction of a regulator	scientific article

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

26 July 2017

Incompetence of neutrophils to invasive group A streptococcus is attributed to induction of plural virulence factors by dysfunction of a regulator (English)

1 reference

Europe PubMed Central

PubMed publication ID

26 July 2017

author name string

Manabu Ato

1

1 reference

Europe PubMed Central

PubMed publication ID

26 July 2017

Tadayoshi Ikebe

2

1 reference

Europe PubMed Central

PubMed publication ID

26 July 2017

Hiroki Kawabata

3

1 reference

Europe PubMed Central

PubMed publication ID

26 July 2017

Toshitada Takemori

4

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Europe PubMed Central

PubMed publication ID

26 July 2017

Haruo Watanabe

5

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Europe PubMed Central

PubMed publication ID

26 July 2017

language of work or name

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publication date

21 October 2008

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Europe PubMed Central

PubMed publication ID

26 July 2017

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Europe PubMed Central

PubMed publication ID

26 July 2017

3

1 reference

Europe PubMed Central

PubMed publication ID

26 July 2017

10

1 reference

Europe PubMed Central

PubMed publication ID

26 July 2017

e3455

1 reference

Europe PubMed Central

PubMed publication ID

26 July 2017

copyright license

Creative Commons Attribution 4.0 International

21 October 2008

1 reference

April 2022 Public Data File from Crossref

copyright status

0 references

A chemokine-degrading extracellular protease made by group A Streptococcus alters pathogenesis by enhancing evasion of the innate immune response

1 reference

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Cirrhosis-induced defects in innate pulmonary defenses against Streptococcus pneumoniae

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A streptococcal protease that degrades CXC chemokines and impairs bacterial clearance from infected tissues

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21 June 2018

How do microbes evade neutrophil killing?

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21 June 2018

Neutrophils and immunity: challenges and opportunities

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DNase expression allows the pathogen group A Streptococcus to escape killing in neutrophil extracellular traps

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21 June 2018

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Effect of a bacterial pheromone peptide on host chemokine degradation in group A streptococcal necrotising soft-tissue infections

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MOTION PICTURE STUDY OF THE TOXIC ACTION OF STREPTOLYSINS ON LEUCOCYTES.

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Bacterial pathogens modulate an apoptosis differentiation program in human neutrophils

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Molecular basis of group A streptococcal virulence

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Cytotoxic effects of streptolysin o and streptolysin s enhance the virulence of poorly encapsulated group a streptococci

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Resealing of large transmembrane pores produced by streptolysin O in nucleated cells is accompanied by NF-kappaB activation and downstream events

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Spontaneous mutations in the CsrRS two-component regulatory system of Streptococcus pyogenes result in enhanced virulence in a murine model of skin and soft tissue infection

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Cytolysin-mediated translocation (CMT): a functional equivalent of type III secretion in gram-positive bacteria

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Inverse relation between disease severity and expression of the streptococcal cysteine protease, SpeB, among clonal M1T1 isolates recovered from invasive group A streptococcal infection cases

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2565068

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Pathogenesis of group A streptococcal infections

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A response regulator that represses transcription of several virulence operons in the group A streptococcus

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Staphylococcal alpha-toxin, streptolysin-O, and Escherichia coli hemolysin: prototypes of pore-forming bacterial cytolysins

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Complement activation and attack on autologous cell membranes induced by streptolysin-O.

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Invasive group A streptococcal infections in Ontario, Canada. Ontario Group A Streptococcal Study Group

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Streptolysin O and adherence synergistically modulate proinflammatory responses of keratinocytes to group A streptococci

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Impairment of polymorphonuclear leukocyte function and metabolic control of diabetes

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Decreased hematopoiesis in bone marrow of mice with congestive heart failure

1 reference

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Increased expression of the ska gene in emm49-genotyped Streptococcus pyogenes strains isolated from patients with severe invasive streptococcal infections

1 reference

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Epidemiological and clinical aspects of invasive group A streptococcal infections and the streptococcal toxic shock syndrome

1 reference

https://pubmed.ncbi.nlm.nih.gov/18941623

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inferred from PubMed ID database lookup

Defining the group A streptococcal toxic shock syndrome. Rationale and consensus definition. The Working Group on Severe Streptococcal Infections

1 reference

https://pubmed.ncbi.nlm.nih.gov/18941623

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

The genetic properties of Streptococcus pyogenes emm49 genotype strains recently emerged among severe invasive infections in Japan

1 reference

https://pubmed.ncbi.nlm.nih.gov/18941623

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

An M protein with a single C repeat prevents phagocytosis of Streptococcus pyogenes: use of a temperature-sensitive shuttle vector to deliver homologous sequences to the chromosome of S. pyogenes

1 reference

https://pubmed.ncbi.nlm.nih.gov/18941623

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inferred from PubMed ID database lookup

Changing prevalent T serotypes and emm genotypes of Streptococcus pyogenes isolates from streptococcal toxic shock-like syndrome (TSLS) patients in Japan

1 reference

https://pubmed.ncbi.nlm.nih.gov/18941623

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1371/JOURNAL.PONE.0003455

1 reference

Europe PubMed Central

PubMed publication ID

26 July 2017

2008PLoSO...3.3455A

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PMC publication ID

1 reference

Europe PubMed Central

PubMed publication ID

26 July 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

26 July 2017

ResearchGate publication ID

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