Mammary epithelial-restricted expression of activated c-src rescues the block to mammary gland morphogenesis due to the deletion of the C-terminus of Patched-1. (Q30010014)

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scientific article published on 9 August 2012

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English	Mammary epithelial-restricted expression of activated c-src rescues the block to mammary gland morphogenesis due to the deletion of the C-terminus of Patched-1.	scientific article published on 9 August 2012

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scholarly article

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Europe PubMed Central

PubMed publication ID

21 May 2017

Mammary epithelial-restricted expression of activated c-src rescues the block to mammary gland morphogenesis due to the deletion of the C-terminus of Patched-1. (English)

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Europe PubMed Central

PubMed publication ID

21 May 2017

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Hong Chang

1

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Europe PubMed Central

PubMed publication ID

21 May 2017

Laurent Balenci

2

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Europe PubMed Central

PubMed publication ID

21 May 2017

Nadia Okolowsky

3

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Europe PubMed Central

PubMed publication ID

21 May 2017

William J Muller

4

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Europe PubMed Central

PubMed publication ID

21 May 2017

Paul A Hamel

5

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Europe PubMed Central

PubMed publication ID

21 May 2017

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publication date

9 August 2012

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Europe PubMed Central

PubMed publication ID

21 May 2017

Developmental Biology

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Europe PubMed Central

PubMed publication ID

21 May 2017

370

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Europe PubMed Central

PubMed publication ID

21 May 2017

2

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Europe PubMed Central

PubMed publication ID

21 May 2017

187-197

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Europe PubMed Central

PubMed publication ID

21 May 2017

Patched1 functions as a gatekeeper by promoting cell cycle progression

1 reference

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The Hedgehog-binding proteins Gas1 and Cdo cooperate to positively regulate Shh signaling during mouse development

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Evaluating Smoothened as a G-protein-coupled receptor for Hedgehog signalling

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Proteolysis That Is Inhibited by Hedgehog Targets Cubitus interruptus Protein to the Nucleus and Converts It to a Repressor

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Mutations in CDON, encoding a hedgehog receptor, result in holoprosencephaly and defective interactions with other hedgehog receptors

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Patched1 interacts with cyclin B1 to regulate cell cycle progression

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Identification of an unexpected link between the Shh pathway and a G2/M regulator, the phosphatase CDC25B

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Mammary gland development and tumorigenesis in estrogen receptor knockout mice

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Noncanonical Hedgehog signaling

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Activation of Erk by sonic hedgehog independent of canonical hedgehog signalling

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Two critical periods of Sonic Hedgehog signaling required for the specification of motor neuron identity

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Conservation of the hedgehog/patched signaling pathway from flies to mice: induction of a mouse patched gene by Hedgehog

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Altered Neural Cell Fates and Medulloblastoma in Mouse patched Mutants

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Protecting the hedgerow: p53 and hedgehog pathway interactions

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Boc and Gas1 each form distinct Shh receptor complexes with Ptch1 and are required for Shh-mediated cell proliferation

1 reference

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Structure-function-folding relationship in a WW domain

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Hedgehog signalling: emerging evidence for non-canonical pathways

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c-Src-null mice exhibit defects in normal mammary gland development and ERalpha signaling

1 reference

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Wnt signal transduction pathways

1 reference

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Insights from the study of animals lacking functional estrogen receptor

1 reference

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Reduced white fat mass in adult mice bearing a truncated Patched 1

1 reference

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Alteration of reproductive function but not prenatal sexual development after insertional disruption of the mouse estrogen receptor gene

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Early and multifocal tumors in breast, salivary, Harderian and epididymal tissues developed in MMTY-Neu transgenic mice

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Structure of the WW domain of a kinase-associated protein complexed with a proline-rich peptide.

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A spontaneous mouse mutation, mesenchymal dysplasia (mes), is caused by a deletion of the most C-terminal cytoplasmic domain of patched (ptc)

1 reference

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Biochemical evidence that patched is the Hedgehog receptor

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Regulation of patched by sonic hedgehog in the developing neural tube

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Hedgehog Pathway Antagonist 5E1 Binds Hedgehog at the Pseudo-active Site

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Activation of the Src/p21ras/Erk pathway by progesterone receptor via cross-talk with estrogen receptor

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Ptch1 is required locally for mammary gland morphogenesis and systemically for ductal elongation

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Constitutive activation of smoothened (SMO) in mammary glands of transgenic mice leads to increased proliferation, altered differentiation and ductal dysplasia

1 reference

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Single-step induction of mammary adenocarcinoma in transgenic mice bearing the activated c-neu oncogene

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Epidermal hyperplasia and expansion of the interfollicular stem cell compartment in mutant mice with a C-terminal truncation of Patched1

1 reference

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Boc is a receptor for sonic hedgehog in the guidance of commissural axons

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Drosophila genome-wide obesity screen reveals hedgehog as a determinant of brown versus white adipose cell fate

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Phage display selection of ligand residues important for Src homology 3 domain binding specificity

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Hedgehog signal transduction: signal integration and cross talk in development and cancer

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Pathways of signal transduction employed by vertebrate Hedgehogs

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GLI3 encodes a 190-kilodalton protein with multiple regions of GLI similarity

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The tumour-suppressor gene patched encodes a candidate receptor for Sonic hedgehog

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Mesenchymal dysplasia: a recessive mutation on chromosome 13 of the mouse

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smoothened encodes a receptor-like serpentine protein required for hedgehog signalling

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Altered differentiation and paracrine stimulation of mammary epithelial cell proliferation by conditionally activated Smoothened

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Promotion of Hras-induced squamous carcinomas by a polymorphic variant of the Patched gene in FVB mice

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Basal Cell Carcinomas Arise from Hair Follicle Stem Cells in Ptch1+/− Mice

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Induction of mammary epithelial hyperplasias and mammary tumors in transgenic mice expressing a murine mammary tumor virus/activated c-src fusion gene

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Sonic hedgehog guides axons through a noncanonical, Src-family-kinase-dependent signaling pathway

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Structural basis for the binding of proline-rich peptides to SH3 domains

1 reference

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Cyclopamine inhibition of human breast cancer cell growth independent of Smoothened (Smo).

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Smoothened mutants reveal redundant roles for Shh and Ihh signaling including regulation of L/R symmetry by the mouse node

1 reference

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10.1016/J.YDBIO.2012.07.027

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