Down-regulation of oxidative phosphorylation in the liver by expression of the ATPase inhibitory factor 1 induces a tumor-promoter metabolic state (Q39509449)

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scientific article published on 22 November 2015

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English	Down-regulation of oxidative phosphorylation in the liver by expression of the ATPase inhibitory factor 1 induces a tumor-promoter metabolic state	scientific article published on 22 November 2015

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1 March 2020

Down-regulation of oxidative phosphorylation in the liver by expression of the ATPase inhibitory factor 1 induces a tumor-promoter metabolic state (English)

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1 March 2020

phosphorylation

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Laura Formentini

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1 March 2020

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Jorgina Satrustegui Gil Delgado

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1 March 2020

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Fulvio Santacatterina

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Laura Sánchez-Cenizo

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Maysa A Mobasher

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Estela Casas

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Inmaculada Martínez-Reyes

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Cristina Núñez de Arenas

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Javier García-Bermúdez

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María Sánchez-Aragó

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Ángela M Valverde

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José M Cuezva

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1 January 2016

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1 March 2020

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1 March 2020

1

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490-508

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A switch in the source of ATP production and a loss in capacity to perform glycolysis are hallmarks of hepatocyte failure in advance liver disease

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Dimers of mitochondrial ATP synthase form the permeability transition pore

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Glucagon regulation of oxidative phosphorylation requires an increase in matrix adenine nucleotide content through Ca2+ activation of the mitochondrial ATP-Mg/Pi carrier SCaMC-3

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Role of the c subunit of the FO ATP synthase in mitochondrial permeability transition

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12 September 2017

Mitochondria-mediated energy adaption in cancer: the H(+)-ATP synthase-geared switch of metabolism in human tumors

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The mitochondrial ATPase inhibitory factor 1 triggers a ROS-mediated retrograde prosurvival and proliferative response

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The AMPK signalling pathway coordinates cell growth, autophagy and metabolism

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