MiR-1 and miR-200 inhibit EMT via Slug-dependent and tumorigenesis via Slug-independent mechanisms (Q39389208)

11 September 2017

title

MiR-1 and miR-200 inhibit EMT via Slug-dependent and tumorigenesis via Slug-independent mechanisms (English)

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11 September 2017

Y-N Liu

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11 September 2017

J J Yin

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11 September 2017

W Abou-Kheir

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11 September 2017

P G Hynes

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11 September 2017

O M Casey

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11 September 2017

L Fang

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11 September 2017

M Yi

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11 September 2017

R M Stephens

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11 September 2017

V Seng

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11 September 2017

H Sheppard-Tillman

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11 September 2017

P Martin

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11 September 2017

K Kelly

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11 September 2017

language of work or name

English

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publication date

27 February 2012

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11 September 2017

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11 September 2017

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11 September 2017

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11 September 2017

page(s)

296-306

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Epithelial-mesenchymal transitions in development and disease

11 September 2017

cites work

1 reference

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A switch from E-cadherin to N-cadherin expression indicates epithelial to mesenchymal transition and is of strong and independent importance for the progress of prostate cancer

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N-cadherin switching occurs in high Gleason grade prostate cancer

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A role for epithelial-mesenchymal transition in the etiology of benign prostatic hyperplasia

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Transforming growth factor-beta promotes invasion in tumorigenic but not in nontumorigenic human prostatic epithelial cells.

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Transforming growth factor beta1 is associated with angiogenesis, metastasis, and poor clinical outcome in prostate cancer

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A reciprocal repression between ZEB1 and members of the miR-200 family promotes EMT and invasion in cancer cells

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The miR-200 family and miR-205 regulate epithelial to mesenchymal transition by targeting ZEB1 and SIP1

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Loss of miR-200 Inhibition of Suz12 Leads to Polycomb-Mediated Repression Required for the Formation and Maintenance of Cancer Stem Cells

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Downregulation of miRNA-200c links breast cancer stem cells with normal stem cells

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Prostate Epithelial Pten/TP53 Loss Leads to Transformation of Multipotential Progenitors and Epithelial to Mesenchymal Transition

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Sarcomatoid carcinoma of the prostate: a study of 42 cases

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Critical and reciprocal regulation of KLF4 and SLUG in transforming growth factor β-initiated prostate cancer epithelial-mesenchymal transition

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The EMT-activator ZEB1 promotes tumorigenicity by repressing stemness-inhibiting microRNAs

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Slug gene expression supports human osteoblast maturation

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Role of Slug transcription factor in human mesenchymal stem cells

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p53 plays a role in mesenchymal differentiation programs, in a cell fate dependent manner

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p53 regulates epithelial-mesenchymal transition and stem cell properties through modulating miRNAs.

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p53 controls cancer cell invasion by inducing the MDM2-mediated degradation of Slug

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Snail2 is an essential mediator of Twist1-induced epithelial mesenchymal transition and metastasis

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Genomic profiling of microRNA and messenger RNA reveals deregulated microRNA expression in prostate cancer

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Methylation mediated silencing of MicroRNA-1 gene and its role in hepatocellular carcinogenesis

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Genome-wide profiling of chromatin signatures reveals epigenetic regulation of MicroRNA genes in colorectal cancer.

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The KinI kinesin Kif2a is required for bipolar spindle assembly through a functional relationship with MCAK

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Lentivirus-mediated bifunctional cell labeling for in vivo melanoma study

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Characterizing the Contribution of Stem/Progenitor Cells to Tumorigenesis in thePten−/−TP53−/− Prostate Cancer Model

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LPA receptor heterodimerizes with CD97 to amplify LPA-initiated RHO-dependent signaling and invasion in prostate cancer cells

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Activation of the RalGEF/Ral pathway promotes prostate cancer metastasis to bone

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Integrated miRNA and mRNA expression profiling of mouse mammary tumor models identifies miRNA signatures associated with mammary tumor lineage

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Elevated levels of circulating interleukin-6 and transforming growth factor-beta1 in patients with metastatic prostatic carcinoma

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PubMed

https://pubmed.ncbi.nlm.nih.gov/22370643

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1038/ONC.2012.58

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Dimensions Publication ID

11 September 2017

1041801251

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PMC publication ID

7580497

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1 reference