Minimal change disease and idiopathic FSGS: manifestations of the same disease. (Q38982416)

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25 March 2020

review article

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title

Minimal change disease and idiopathic FSGS: manifestations of the same disease (English)

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25 March 2020

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25 March 2020

Rutger J Maas

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25 March 2020

Jeroen K Deegens

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25 March 2020

Marcus J Moeller

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25 March 2020

Jack F Wetzels

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25 March 2020

language of work or name

English

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publication date

17 October 2016

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Nature Reviews Nephrology

25 March 2020

published in

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25 March 2020

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25 March 2020

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25 March 2020

page(s)

768-776

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Causes and pathogenesis of focal segmental glomerulosclerosis

25 March 2020

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Detection of activated parietal epithelial cells on the glomerular tuft distinguishes early focal segmental glomerulosclerosis from minimal change disease

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Associated focal and segmental glomerulosclerosis in the acquired immunodeficiency syndrome.

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A spectrum of morphologic lesions of focal segmental glomerulosclerosis by Columbia criteria in human immunodeficiency virus infection

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The Mechanism of Proteinuria

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The renal functional and structural consequences of corticosteroid and angiotensin-converting enzyme inhibitor therapy in chronic puromycin aminonucleoside nephropathy

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Dexamethasone prevents podocyte apoptosis induced by puromycin aminonucleoside: role of p53 and Bcl-2-related family proteins

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Podocyte-secreted angiopoietin-like-4 mediates proteinuria in glucocorticoid-sensitive nephrotic syndrome

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Aminopeptidase A: a nephritogenic target antigen of nephrotoxic serum

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Circulating Permeability Factors in Idiopathic Nephrotic Syndrome and Focal Segmental Glomerulosclerosis

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T-cell transcriptome analysis points up a thymic disorder in idiopathic nephrotic syndrome.

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Altered activity of plasma hemopexin in patients with minimal change disease in relapse

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Serum-soluble urokinase receptor levels do not distinguish focal segmental glomerulosclerosis from other causes of nephrotic syndrome in children

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A reassessment of soluble urokinase-type plasminogen activator receptor in glomerular disease

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Pathogenesis of lipoid nephrosis: a disorder of T-cell function

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T regulatory cell function in idiopathic minimal lesion nephrotic syndrome.

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Induction of T regulatory cells attenuates idiopathic nephrotic syndrome

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Upregulation of nuclear factor-related kappa B suggests a disorder of transcriptional regulation in minimal change nephrotic syndrome

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21 January 2018

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CD80 and suPAR in patients with minimal change disease and focal segmental glomerulosclerosis: diagnostic and pathogenic significance

21 January 2018

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Abatacept in B7-1-positive proteinuric kidney disease

21 January 2018

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Any value of podocyte B7-1 as a biomarker in human MCD and FSGS?

21 January 2018

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B7-1 Blockade Does Not Improve Post-Transplant Nephrotic Syndrome Caused by Recurrent FSGS.

21 January 2018

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Case series: CTLA4-IgG1 therapy in minimal change disease and focal segmental glomerulosclerosis

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A molecular profile of focal segmental glomerulosclerosis from formalin-fixed, paraffin-embedded tissue

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Podocyte Injury Damages Other Podocytes

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12 December 2020

Plasma exchange improves graft survival in patients with recurrent focal glomerulosclerosis after renal transplant

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A hitherto undescribed vulnerability of the juxtamedullary glomeruli in lipoid nephrosis

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Focal and segmental glomerulosclerosis following a single intravenous dose of puromycin aminonucleoside

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Recurrent focal segmental glomerulosclerosis in the renal allograft: single center experience in the era of modern immunosuppression

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12 December 2020

Identifiers

DOI

10.1038/NRNEPH.2016.147

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:27748392%20AND%20SRC:MED&resulttype=core&format=json

25 March 2020

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:27748392%20AND%20SRC:MED&resulttype=core&format=json