Expression of Fas ligand in activated T cells is regulated by c-Myc (Q38313969)

2 September 2017

title

Expression of Fas ligand in activated T cells is regulated by c-Myc (English)

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2 September 2017

Brunner T

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2 September 2017

Kasibhatla S

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2 September 2017

Pinkoski MJ

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2 September 2017

Frutschi C

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2 September 2017

Yoo NJ

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2 September 2017

Echeverri F

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2 September 2017

Mahboubi A

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2 September 2017

Green DR

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2 September 2017

language of work or name

English

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publication date

1 March 2000

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Journal of Biological Chemistry

2 September 2017

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inferred from page(s)

published in

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2 September 2017

volume

275

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2 September 2017

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2 September 2017

page(s)

9767-9772

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Induction of apoptosis in fibroblasts by c-myc protein

2 September 2017

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Role for c-myc in activation-induced apoptotic cell death in T cell hybridomas

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Functional Myc-Max heterodimer is required for activation-induced apoptosis in T cell hybridomas.

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Fas ligand mediates activation-induced cell death in human T lymphocytes

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Requirement for the CD95 Receptor-Ligand Pathway in c-Myc-Induced Apoptosis

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DNA damaging agents induce expression of Fas ligand and subsequent apoptosis in T lymphocytes via the activation of NF-kappa B and AP-1.

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Identification of a monoclonal antibody specific for a murine T3 polypeptide

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Human Fas ligand: gene structure, chromosomal location and species specificity

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Promoter independent down-regulation of the firefly luciferase gene by T3 and T3 receptor in CV1 cells

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Myc and Max proteins possess distinct transcriptional activities

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Inducible nonlymphoid expression of Fas ligand is responsible for superantigen-induced peripheral deletion of T cells

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Transforming growth factor beta1 inhibits Fas ligand expression and subsequent activation-induced cell death in T cells via downregulation of c-Myc

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Control of CD4 effector fate: transforming growth factor beta 1 and interleukin 2 synergize to prevent apoptosis and promote effector expansion

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Regulation of fas-ligand expression during activation-induced cell death in T lymphocytes via nuclear factor kappaB.

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The ornithine decarboxylase gene is a transcriptional target of c-Myc

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Increased expression of eukaryotic translation initiation factors eIF-4E and eIF-2 alpha in response to growth induction by c-myc

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An essential E box in the promoter of the gene encoding the mRNA cap-binding protein (eukaryotic initiation factor 4E) is a target for activation by c-myc

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Ornithine decarboxylase activity is critical for cell transformation

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Ornithine decarboxylase is a mediator of c-Myc-induced apoptosis

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Cdc25 cell-cycle phosphatase as a target of c-myc

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The Fas counterattack: Fas-mediated T cell killing by colon cancer cells expressing Fas ligand

21 January 2018

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CD95 ligand in graft rejection

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21 January 2018

Identifiers

DOI

10.1074/JBC.275.13.9767

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