The sandy (sdy) mouse: a dysbindin-1 mutant relevant to schizophrenia research (Q37714973)

22 August 2017

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title

The sandy (sdy) mouse: a dysbindin-1 mutant relevant to schizophrenia research (English)

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22 August 2017

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Konrad Talbot

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22 August 2017

publication date

20 November 2009

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Progress in Brain Research

22 August 2017

published in

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22 August 2017

volume

179

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22 August 2017

page(s)

87-94

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Systematic meta-analyses and field synopsis of genetic association studies in schizophrenia: the SzGene database

22 August 2017

cites work

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Mutations in genes encoding melanosomal proteins cause pigmentary glaucoma in DBA/2J mice

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Dysbindin, a novel coiled-coil-containing protein that interacts with the dystrobrevins in muscle and brain

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Behavioral characterization of dysbindin-1 deficient sandy mice

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DTNBP1, a schizophrenia susceptibility gene, affects kinetics of transmitter release

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Neurobehavioral abnormalities in the dysbindin-1 mutant, sandy, on a C57BL/6J genetic background

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Dysbindin deficiency in sandy mice causes reduction of snapin and displays behaviors related to schizophrenia

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Spatial working memory as an endophenotype for schizophrenia

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Impairment on the hippocampal-dependent virtual Morris water task in schizophrenia

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Behavioral abnormalities and dopamine reductions in sdy mutant mice with a deletion in Dtnbp1, a susceptibility gene for schizophrenia.

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Evidence that the BLOC-1 protein dysbindin modulates dopamine D2 receptor internalization and signaling but not D1 internalization

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Dysbindin modulates prefrontal cortical glutamatergic circuits and working memory function in mice.

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Hyperactivation of midbrain dopaminergic system in schizophrenia could be attributed to the down-regulation of dysbindin.

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Cognitive dysfunction in schizophrenia: convergence of gamma-aminobutyric acid and glutamate alterations

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Hermansky-Pudlak syndrome type 7 (HPS-7) results from mutant dysbindin, a member of the biogenesis of lysosome-related organelles complex 1 (BLOC-1)

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Can the role of genetic factors in schizophrenia be enlightened by studies of candidate gene mutant mice behaviour?

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High dopamine turnover in the brains of Sandy mice.

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Evidence of novel neuronal functions of dysbindin, a susceptibility gene for schizophrenia

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Snapin facilitates the synchronization of synaptic vesicle fusion

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Schizophrenia-relevant behavioral testing in rodent models: a uniquely human disorder?

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BLOC-1 is required for cargo-specific sorting from vacuolar early endosomes toward lysosome-related organelles

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Genetic variation in the 6p22.3 gene DTNBP1, the human ortholog of the mouse dysbindin gene, is associated with schizophrenia

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Candidate genes for schizophrenia: a survey of association studies and gene ranking

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Sandy: a new mouse model for platelet storage pool deficiency

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Impaired long-term memory retention and working memory in sdy mutant mice with a deletion in Dtnbp1, a susceptibility gene for schizophrenia

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7 January 2021

Dysbindin-1 is a synaptic and microtubular protein that binds brain snapin

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7 January 2021

Dysbindin-1 is reduced in intrinsic, glutamatergic terminals of the hippocampal formation in schizophrenia

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Dysbindin-1 in dorsolateral prefrontal cortex of schizophrenia cases is reduced in an isoform-specific manner unrelated to dysbindin-1 mRNA expression

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The role of Snapin in neurosecretion: snapin knock-out mice exhibit impaired calcium-dependent exocytosis of large dense-core vesicles in chromaffin cells

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Identifiers

DOI

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22 August 2017

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