Pathogenic Transdifferentiation of Th17 Cells Contribute to Perpetuation of Rheumatoid Arthritis during Anti-TNF Treatment (Q36167063)

12 August 2017

Pathogenic Transdifferentiation of Th17 Cells Contribute to Perpetuation of Rheumatoid Arthritis during Anti-TNF Treatment (English)

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12 August 2017

0 references

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Ron Cialic

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Rille Pullerits

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Hadi Valadi

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Malin C Erlandsson

1 reference

12 August 2017

Howard L Weiner

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12 August 2017

Vijay K Kuchroo

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12 August 2017

Karin M E Andersson

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12 August 2017

Nicola Filluelo Cavallini

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12 August 2017

Dan Hu

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12 August 2017

Mikael Brisslert

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12 August 2017

Sofia Silfverswärd

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12 August 2017

Maria I Bokarewa

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12 August 2017

language of work or name

English

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publication date

4 June 2015

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12 August 2017

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12 August 2017

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12 August 2017

536-543

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Creative Commons Attribution 4.0 International

12 August 2017

start time

1 January 2015

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April 2022 Public Data File from Crossref

copyrighted

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cites work

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SOCS1 is essential for regulatory T cell functions by preventing loss of Foxp3 expression as well as IFN-{gamma} and IL-17A production

1 October 2017

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Increasing levels of circulating Th17 cells and interleukin-17 in rheumatoid arthritis patients with an inadequate response to anti-TNF-α therapy

1 October 2017

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Human RORγt+ TH17 cells preferentially differentiate from naive FOXP3+Treg in the presence of lineage-specific polarizing factors

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1 October 2017

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1 October 2017

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1 October 2017

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IL-17-producing human peripheral regulatory T cells retain suppressive function

1 October 2017

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1 October 2017

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1 October 2017

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1 October 2017

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1 October 2017

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1 October 2017

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1 October 2017

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1 October 2017

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1 October 2017

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1 October 2017

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The american rheumatism association 1987 revised criteria for the classification of rheumatoid arthritis

1 October 2017

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A phase II randomized study of subcutaneous ixekizumab, an anti-interleukin-17 monoclonal antibody, in rheumatoid arthritis patients who were naive to biologic agents or had an inadequate response to tumor necrosis factor inhibitors

1 October 2017

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Pathogenic conversion of Foxp3+ T cells into TH17 cells in autoimmune arthritis

23 September 2018

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Human memory Helios- FOXP3+ regulatory T cells (Tregs) encompass induced Tregs that express Aiolos and respond to IL-1β by downregulating their suppressor functions

23 September 2018

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Efficacy and safety of secukinumab in patients with rheumatoid arthritis: a phase II, dose-finding, double-blind, randomised, placebo controlled study

23 September 2018

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Interleukin-1 and IL-23 induce innate IL-17 production from gammadelta T cells, amplifying Th17 responses and autoimmunity.

23 September 2018

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Human CD25highFoxp3pos regulatory T cells differentiate into IL-17-producing cells

23 September 2018

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CCR6 expression defines regulatory effector/memory-like cells within the CD25(+)CD4+ T-cell subset.

23 September 2018

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Tumor necrosis factor-interleukin-17 interplay induces S100A8, interleukin-1β, and matrix metalloproteinases, and drives irreversible cartilage destruction in murine arthritis: Rationale for combination treatment during arthritis

23 September 2018

2 references

https://api.crossref.org/works/10.2119%2FMOLMED.2015.00057

31 March 2020

https://pubmed.ncbi.nlm.nih.gov/26062018

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Brief report: inhibition of interleukin-6 function corrects Th17/Treg cell imbalance in patients with rheumatoid arthritis

1 reference

https://api.crossref.org/works/10.2119%2FMOLMED.2015.00057

Th17 cells are restrained by Treg cells via the inhibition of interleukin-6 in patients with rheumatoid arthritis responding to anti-tumor necrosis factor antibody therapy

31 March 2020

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Seventeen-colour flow cytometry: unravelling the immune system

31 March 2020

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Serum IL-6 and IL-21 are associated with markers of B cell activation and structural progression in early rheumatoid arthritis: results from the ESPOIR cohort

31 March 2020

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10.2119/MOLMED.2015.00057

31 March 2020

Identifiers

DOI

1 reference

Dimensions Publication ID

12 August 2017

0 references

1 reference

12 August 2017

PubMed publication ID

26062018

1 reference