Neurodegeneration: how does parkin prevent Parkinson's disease? (Q35169544)

6 August 2017

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title

Neurodegeneration: how does parkin prevent Parkinson's disease? (English)

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6 August 2017

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Mark R Cookson

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6 August 2017

language of work or name

English

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publication date

1 July 2003

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6 August 2017

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6 August 2017

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6 August 2017

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6 August 2017

page(s)

R522-4

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6 August 2017

cites work

Genes and parkinsonism

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https://api.crossref.org/works/10.1016%2FS0960-9822%2803%2900446-9

7 January 2021

The lore of the RINGs: substrate recognition and catalysis by ubiquitin ligases

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https://api.crossref.org/works/10.1016%2FS0960-9822%2803%2900446-9

7 January 2021

Parkin suppresses dopaminergic neuron-selective neurotoxicity induced by Pael-R in Drosophila

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2803%2900446-9

7 January 2021

Parkin protects against the toxicity associated with mutant alpha-synuclein: proteasome dysfunction selectively affects catecholaminergic neurons

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2803%2900446-9

7 January 2021

An unfolded putative transmembrane polypeptide, which can lead to endoplasmic reticulum stress, is a substrate of Parkin

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2803%2900446-9

7 January 2021

Parkin is a component of an SCF-like ubiquitin ligase complex and protects postmitotic neurons from kainate excitotoxicity

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2803%2900446-9

7 January 2021

Parkin prevents mitochondrial swelling and cytochrome c release in mitochondria-dependent cell death.

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2803%2900446-9

7 January 2021

Parkin and CASK/LIN-2 associate via a PDZ-mediated interaction and are co-localized in lipid rafts and postsynaptic densities in brain

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2803%2900446-9

7 January 2021

Culprits in the degradation of cyclin E apprehended

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https://api.crossref.org/works/10.1016%2FS0960-9822%2803%2900446-9

7 January 2021

Parkin binds the Rpn10 subunit of 26S proteasomes through its ubiquitin-like domain

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https://api.crossref.org/works/10.1016%2FS0960-9822%2803%2900446-9

7 January 2021

Ubiquitination of a new form of alpha-synuclein by parkin from human brain: implications for Parkinson's disease

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2803%2900446-9

7 January 2021

Molecular crowding accelerates fibrillization of alpha-synuclein: could an increase in the cytoplasmic protein concentration induce Parkinson's disease?

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2803%2900446-9

7 January 2021

Dopamine-dependent neurotoxicity of alpha-synuclein: a mechanism for selective neurodegeneration in Parkinson disease

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https://api.crossref.org/works/10.1016%2FS0960-9822%2803%2900446-9

7 January 2021

Acceleration of oligomerization, not fibrillization, is a shared property of both alpha-synuclein mutations linked to early-onset Parkinson's disease: implications for pathogenesis and therapy

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2803%2900446-9

7 January 2021

Inducible expression of mutant alpha-synuclein decreases proteasome activity and increases sensitivity to mitochondria-dependent apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2803%2900446-9

7 January 2021

Mitochondrial pathology and apoptotic muscle degeneration in Drosophila parkin mutants

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2803%2900446-9

7 January 2021

10.1016/S0960-9822(03)00446-9

Identifiers

DOI

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6 August 2017

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