High serum endostatin levels in Down syndrome: implications for improved treatment and prevention of solid tumours (Q34108448)

31 July 2017

title

High serum endostatin levels in Down syndrome: implications for improved treatment and prevention of solid tumours (English)

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31 July 2017

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Zatz M

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31 July 2017

Zorick TS

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31 July 2017

Mustacchi Z

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31 July 2017

Bando SY

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31 July 2017

Moreira-Filho CA

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31 July 2017

Olsen B

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31 July 2017

Passos-Bueno MR

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31 July 2017

publication date

1 November 2001

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European Journal of Human Genetics

31 July 2017

published in

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31 July 2017

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31 July 2017

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31 July 2017

page(s)

811-814

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Risks of leukaemia and solid tumours in individuals with Down's syndrome.

31 July 2017

cites work

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Mouse Col18a1 is expressed in a tissue-specific manner as three alternative variants and is localized in basement membrane zones

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Adenovirus-mediated gene transfer of endostatin in vivo results in high level of transgene expression and inhibition of tumor growth and metastases

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The short and long forms of type XVIII collagen show clear tissue specificities in their expression and location in basement membrane zones in humans

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Collagen XVIII, containing an endogenous inhibitor of angiogenesis and tumor growth, plays a critical role in the maintenance of retinal structure and in neural tube closure (Knobloch syndrome)

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Secreted cathepsin L generates endostatin from collagen XVIII

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Structure, function and tissue forms of the C-terminal globular domain of collagen XVIII containing the angiogenesis inhibitor endostatin

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Increased concentrations of the circulating angiogenesis inhibitor endostatin in patients with systemic sclerosis

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Intracellular Ca(2+) signaling in endothelial cells by the angiogenesis inhibitors endostatin and angiostatin

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Cell surface glypicans are low-affinity endostatin receptors

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Identifiers

DOI

10.1038/SJ.EJHG.5200721

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