Alzheimer's disease-do tauists and baptists finally shake hands? (Q28215968)

https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Genes, models and Alzheimer's disease

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Intracellular APP processing and A beta production in Alzheimer disease

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Targeting small Abeta oligomers: the solution to an Alzheimer's disease conundrum?

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

A transcriptionally [correction of transcriptively] active complex of APP with Fe65 and histone acetyltransferase Tip60

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Susceptibility locus for Alzheimer's disease on chromosome 10.

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Linkage of plasma Abeta42 to a quantitative locus on chromosome 10 in late-onset Alzheimer's disease pedigrees

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Activation of tau protein kinase I/glycogen synthase kinase-3beta by amyloid beta peptide (25-35) enhances phosphorylation of tau in hippocampal neurons

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Tau protein kinase I is essential for amyloid beta-protein-induced neurotoxicity

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Lithium protects cultured neurons against beta-amyloid-induced neurodegeneration

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Mutant presenilins of Alzheimer's disease increase production of 42-residue amyloid beta-protein in both transfected cells and transgenic mice

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Increased amyloid-beta42(43) in brains of mice expressing mutant presenilin 1

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

APPSW Transgenic Mice Develop Age-related Aβ Deposits and Neuropil Abnormalities, but no Neuronal Loss in CA1

1 reference

https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Accelerated Alzheimer-type phenotype in transgenic mice carrying both mutant amyloid precursor protein and presenilin 1 transgenes

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor protein

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Peripherally administered antibodies against amyloid beta-peptide enter the central nervous system and reduce pathology in a mouse model of Alzheimer disease

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

A beta peptide vaccination prevents memory loss in an animal model of Alzheimer's disease

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Immunization with amyloid-beta attenuates Alzheimer-disease-like pathology in the PDAPP mouse

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Alzheimer's disease: paired helical filaments and cytomembranes

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Staging the pathological assembly of truncated tau protein into paired helical filaments in Alzheimer's disease.

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Phosphorylation that detaches tau protein from microtubules (Ser262, Ser214) also protects it against aggregation into Alzheimer paired helical filaments

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Evolution of neuronal changes in the course of Alzheimer's disease.

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

A sequence of cytoskeleton changes related to the formation of neurofibrillary tangles and neuropil threads

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Correlation between elevated levels of amyloid beta-peptide in the brain and cognitive decline

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Untangling tau-related dementia

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Mutation-dependent aggregation of tau protein and its selective depletion from the soluble fraction in brain of P301L FTDP-17 patients

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Polymerization of tau peptides into fibrillar structures. The effect of FTDP-17 mutations

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

The FTDP-17-linked mutation R406W abolishes the interaction of phosphorylated tau with microtubules

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Effects of frontotemporal dementia FTDP-17 mutations on heparin-induced assembly of tau filaments

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Accelerated filament formation from tau protein with specific FTDP-17 missense mutations.

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Tau proteins with FTDP-17 mutations have a reduced ability to promote microtubule assembly

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Mutations in tau reduce its microtubule binding properties in intact cells and affect its phosphorylation

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Missense and silent tau gene mutations cause frontotemporal dementia with parkinsonism-chromosome 17 type, by affecting multiple alternative RNA splicing regulatory elements

1 reference

https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

5' splice site mutations in tau associated with the inherited dementia FTDP-17 affect a stem-loop structure that regulates alternative splicing of exon 10.

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

FTDP-17 mutations N279K and S305N in tau produce increased splicing of exon 10

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Fibrillogenesis of tau: insights from tau missense mutations in FTDP-17.

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Competition for microtubule-binding with dual expression of tau missense and splice isoforms

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Enhanced neurofibrillary degeneration in transgenic mice expressing mutant tau and APP.

1 reference

https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Formation of neurofibrillary tangles in P301l tau transgenic mice induced by Abeta 42 fibrils

1 reference

https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Age-Dependent Emergence and Progression of a Tauopathy in Transgenic Mice Overexpressing the Shortest Human Tau Isoform

1 reference

https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Age-dependent induction of congophilic neurofibrillary tau inclusions in tau transgenic mice

1 reference

https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Coexisting pathologies in the brain: influence of vascular disease and Parkinson's disease on Alzheimer's pathology in the hippocampus.

1 reference

https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Are Alzheimer's disease, hypertension, and cerebrocapillary damage related?.

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

The role of cerebral ischemia in Alzheimer's disease

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Does dysregulation of the Notch and wingless/Wnt pathways underlie the pathogenesis of Alzheimer's disease?

1 reference

https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Wnt signaling function in Alzheimer's disease

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https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Presenilin 1 negatively regulates beta-catenin/T cell factor/lymphoid enhancer factor-1 signaling independently of beta-amyloid precursor protein and notch processing

1 reference

https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Presenilins interact with armadillo proteins including neural-specific plakophilin-related protein and beta-catenin

1 reference

https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Presenilin 1 associates with glycogen synthase kinase-3beta and its substrate tau

1 reference

https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Human presenilin-1, but not familial Alzheimer's disease (FAD) mutants, facilitate Caenorhabditis elegans Notch signalling independently of proteolytic processing

1 reference

https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

A presenilin-1-dependent gamma-secretase-like protease mediates release of Notch intracellular domain

1 reference

https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Interaction between Wingless and Notch signaling pathways mediated by dishevelled

1 reference

https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

Dishevelled regulates the metabolism of amyloid precursor protein via protein kinase C/mitogen-activated protein kinase and c-Jun terminal kinase.

1 reference

https://api.crossref.org/works/10.1016%2FS0166-2236%2800%2902031-2

7 January 2021

10.1016/S0166-2236(00)02031-2

Identifiers

DOI

1 reference

Consolidated OpenCitations Corpus – April 2017

1 reference

Consolidated OpenCitations Corpus – April 2017

PubMed publication ID

11801334

1 reference

Consolidated OpenCitations Corpus – April 2017