Epigenetic silencing of the HIC-1 gene in human medulloblastomas (Q28189231)

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Andreas Waha

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Arend Koch

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Birgit Meyer-Puttlitz

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Sascha Weggen

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Nils Sörensen

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Steffen Albrecht

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Cynthia G Goodyer

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Frank Berthold

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Otmar D Wiestler

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Torsten Pietsch

Journal of Neuropathology & Experimental Neurology

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language of work or name

English

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publication date

November 2003

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published in

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volume

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issue

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page(s)

1192-201

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cites work

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Molecular analysis of the lissencephaly gene 1 (LIS-1) in medulloblastomas

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p53 activates expression of HIC-1, a new candidate tumour suppressor gene on 17p13.3

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Recruitment of SMRT/N-CoR-mSin3A-HDAC-repressing complexes is not a general mechanism for BTB/POZ transcriptional repressors: the case of HIC-1 and gammaFBP-B

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Identification in the human candidate tumor suppressor gene HIC-1 of a new major alternative TATA-less promoter positively regulated by p53

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Isolation of a Miller-Dieker lissencephaly gene containing G protein beta-subunit-like repeats

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Mice deficient in the candidate tumor suppressor gene Hic1 exhibit developmental defects of structures affected in the Miller-Dieker syndrome.

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Heterozygous disruption of Hic1 predisposes mice to a gender-dependent spectrum of malignant tumors

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Methylation of the HIC-1 candidate tumor suppressor gene in human breast cancer

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DNA hypermethylation at the D17S5 locus and reduced HIC-1 mRNA expression are associated with hepatocarcinogenesis.

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Microsatellite analysis of loss of heterozygosity on chromosomes 9q, 11p and 17p in medulloblastomas

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CpG islands and genes

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Aberrant CpG-island methylation has non-random and tumour-type-specific patterns.

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Aberrant promoter methylation of previously unidentified target genes is a common abnormality in medulloblastomas--implications for tumor biology and potential clinical utility.

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Methylation, mutation and cancer

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