c-Src regulates Akt signaling in response to ghrelin via beta-arrestin signaling-independent and -dependent mechanisms (Q24317611)

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English	c-Src regulates Akt signaling in response to ghrelin via beta-arrestin signaling-independent and -dependent mechanisms	scientific article

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scholarly article

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title

c-Src regulates Akt signaling in response to ghrelin via beta-arrestin signaling-independent and -dependent mechanisms (English)

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author

Marily Theodoropoulou

series ordinal

2

object named as

Marily Theodoropoulou

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Maria Lodeiro

object named as

Maria Lodeiro

series ordinal

1

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María Pardo

object named as

Maria Pardo

series ordinal

3

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author name string

Felipe F Casanueva

series ordinal

4

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Jesus P Camiña

series ordinal

5

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language of work or name

English

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publication date

2009

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published in

PLOS One

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volume

4

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issue

3

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page(s)

e4686

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copyright license

Creative Commons Attribution 4.0 International

start time

5 March 2009

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stated in

April 2022 Public Data File from Crossref

copyright status

copyrighted

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cites work

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PHLPP and a second isoform, PHLPP2, differentially attenuate the amplitude of Akt signaling by regulating distinct Akt isoforms

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Ghrelin enhances the proliferating effect of thyroid stimulating hormone in FRTL-5 thyroid cells

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Stimulation by ghrelin of p42/p44 mitogen-activated protein kinase through the GHS-R1a receptor: role of G-proteins and beta-arrestins

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Role of beta-arrestin 1 in the metastatic progression of colorectal cancer

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Interaction between Src and a C-terminal proline-rich motif of Akt is required for Akt activation

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β-Arrestin1 Mediates Insulin-like Growth Factor 1 (IGF-1) Activation of Phosphatidylinositol 3-Kinase (PI3K) and Anti-apoptosis

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Direct identification of tyrosine 474 as a regulatory phosphorylation site for the Akt protein kinase

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Ghrelin enhances in vivo skeletal muscle but not liver AKT signaling in rats

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Octreotide, a somatostatin analogue, mediates its antiproliferative action in pituitary tumor cells by altering phosphatidylinositol 3-kinase signaling and inducing Zac1 expression

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Ghrelin has novel vascular actions that mimic PI 3-kinase-dependent actions of insulin to stimulate production of NO from endothelial cells

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Ghrelin inhibits angiotensin II-induced migration of human aortic endothelial cells

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based on heuristic

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Effect of ghrelin on human endothelial cells apoptosis induced by high glucose

1 reference

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reference URL

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based on heuristic

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alpha-Thrombin induces rapid and sustained Akt phosphorylation by beta-arrestin1-dependent and -independent mechanisms, and only the sustained Akt phosphorylation is essential for G1 phase progression

1 reference

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reference URL

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based on heuristic

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Identifiers

DOI

10.1371/JOURNAL.PONE.0004686

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

3778193

ADS bibcode

2009PLoSO...4.4686L

0 references

OpenCitations bibliographic resource ID

3778193

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

3778193

PMC publication ID

2650409

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

3778193

PubMed publication ID

19262695

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

3778193

ResearchGate publication ID

24178601

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