Pages that link to "Q37116056"
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The following pages link to RIPK1 and RIPK3 Kinases Promote Cell-Death-Independent Inflammation by Toll-like Receptor 4. (Q37116056):
Displaying 50 items.
- Activity of Uncleaved Caspase-8 Controls Anti-bacterial Immune Defense and TLR-Induced Cytokine Production Independent of Cell Death (Q36162984) (← links)
- Genetic Inhibition of Receptor Interacting Protein Kinase-1 Reduces Cell Death and Improves Functional Outcome After Intracerebral Hemorrhage in Mice (Q38648036) (← links)
- Participation of Necroptosis in the Host Response to Acute Bacterial Pneumonia (Q38765493) (← links)
- Recent early clinical drug development for acute kidney injury. (Q38780169) (← links)
- RIPK3 Restricts Viral Pathogenesis via Cell Death-Independent Neuroinflammation (Q38862204) (← links)
- Cell death-independent activities of the death receptors CD95, TRAILR1, and TRAILR2. (Q39013916) (← links)
- Controlled detonation: evolution of necroptosis in pathogen defense. (Q39026526) (← links)
- Relevance of necroptosis in cancer (Q39030578) (← links)
- Necroptosis in development, inflammation and disease. (Q39044997) (← links)
- Complex Pathologic Roles of RIPK1 and RIPK3: Moving Beyond Necroptosis (Q39105381) (← links)
- RIPK3 in cell death and inflammation: the good, the bad, and the ugly. (Q39277433) (← links)
- Necroptosis in cancer: An angel or a demon? (Q39398933) (← links)
- RIPK1-dependent apoptosis bypasses pathogen blockade of innate signaling to promote immune defense. (Q40074113) (← links)
- Host response: Neurons loosen the gRIP of death (Q40203808) (← links)
- Kinase Activities of RIPK1 and RIPK3 Can Direct IFN-β Synthesis Induced by Lipopolysaccharide. (Q40219746) (← links)
- The role of major virulence factors of AIEC involved in inflammatory bowl disease-a mini-review. (Q41997378) (← links)
- Necroptosis Execution Is Mediated by Plasma Membrane Nanopores Independent of Calcium (Q42210642) (← links)
- The caspase-8/RIPK3 signaling axis in antigen presenting cells controls the inflammatory arthritic response. (Q42278840) (← links)
- Necroptosis controls NET generation and mediates complement activation, endothelial damage, and autoimmune vasculitis (Q47594489) (← links)
- Necroptotic cell death in anti-cancer therapy (Q47659073) (← links)
- Immunological consequences of kidney cell death (Q49679688) (← links)
- RIP kinase 1-dependent endothelial necroptosis underlies systemic inflammatory response syndrome. (Q52718845) (← links)
- Constitutive interferon signaling maintains critical threshold of MLKL expression to license necroptosis. (Q54108143) (← links)
- Caspase-8 deficiency in mouse embryos triggers chronic RIPK1-dependent activation of inflammatory genes, independently of RIPK3. (Q54115576) (← links)
- Is Receptor-Interacting Protein Kinase 3 a Viable Therapeutic Target for Mycobacterium tuberculosis Infection? (Q54976471) (← links)
- Plasmatic membrane toll-like receptor expressions in human astrocytomas. (Q55279509) (← links)
- Swine Influenza Virus Induces RIPK1/DRP1-Mediated Interleukin-1 Beta Production (Q57005582) (← links)
- Comparing the effects of different cell death programs in tumor progression and immunotherapy (Q57809694) (← links)
- BRAF and AXL oncogenes drive RIPK3 expression loss in cancer (Q58696646) (← links)
- RIPK1 is a critical modulator of both tonic and TLR-responsive inflammatory and cell death pathways in human macrophage differentiation (Q58700548) (← links)
- TNFR2 unlocks a RIPK1 kinase activity-dependent mode of proinflammatory TNFR1 signaling (Q58751569) (← links)
- Necroptosis mediates myofibre death in dystrophin-deficient mice (Q58759838) (← links)
- RIPK1-RIPK3-MLKL-Associated Necroptosis Drives Killing in Neutrophils (Q58781513) (← links)
- c-Jun N-terminal kinases differentially regulate TNF- and TLRs-mediated necroptosis through their kinase-dependent and -independent activities (Q59136662) (← links)
- Necroptosis in anti-viral inflammation (Q59355930) (← links)
- Necrostatin-1 Attenuates Trauma-Induced Mouse Osteoarthritis and IL-1β Induced Apoptosis via HMGB1/TLR4/SDF-1 in Primary Mouse Chondrocytes (Q59807296) (← links)
- Complement Factor H and Apolipoprotein E Participate in Regulation of Inflammation in THP-1 Macrophages (Q59813473) (← links)
- BID-ding on necroptosis in MDS (Q60912334) (← links)
- Recent advances in understanding inhibitor of apoptosis proteins (Q60920665) (← links)
- RIPK3-Mediated Necroptosis and Neutrophil Infiltration Are Associated with Poor Prognosis in Patients with Alcoholic Cirrhosis (Q60948076) (← links)
- Targeting RIPK1 for the treatment of human diseases (Q64265295) (← links)
- Neuronal Cell Death (Q87906376) (← links)
- Tristetraprolin regulates necroptosis during tonic Toll-like receptor 4 (TLR4) signaling in murine macrophages (Q89865303) (← links)
- High mobility group box 1 enables bacterial lipids to trigger receptor-interacting protein kinase 3 (RIPK3)-mediated necroptosis and apoptosis in mice (Q89997813) (← links)
- TNFR2 induced priming of the inflammasome leads to a RIPK1-dependent cell death in the absence of XIAP (Q90196931) (← links)
- Biallelic RIPK1 mutations in humans cause severe immunodeficiency, arthritis, and intestinal inflammation (Q90349450) (← links)
- Decreased RIPK1 expression in chondrocytes alleviates osteoarthritis via the TRIF/MyD88-RIPK1-TRAF2 negative feedback loop (Q90673098) (← links)
- RIP kinases as modulators of inflammation and immunity (Q91069815) (← links)
- Necroptosis is dispensable for motor neuron degeneration in a mouse model of ALS (Q91345013) (← links)
- O-GlcNAc Transferase Suppresses Inflammation and Necroptosis by Targeting Receptor-Interacting Serine/Threonine-Protein Kinase 3 (Q91631677) (← links)