Pages that link to "Q37037953"
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The following pages link to Mitochondria in the aetiology and pathogenesis of Parkinson's disease (Q37037953):
Displaying 50 items.
- PINK1 is selectively stabilized on impaired mitochondria to activate Parkin (Q21145802) (← links)
- Mitochondrial dysfunction and loss of Parkinson's disease-linked proteins contribute to neurotoxicity of manganese-containing welding fumes (Q23916368) (← links)
- Functional alteration of PARL contributes to mitochondrial dysregulation in Parkinson's disease (Q24294218) (← links)
- Parkin interacts with Klokin1 for mitochondrial import and maintenance of membrane potential (Q24297216) (← links)
- The mitochondrial intramembrane protease PARL cleaves human Pink1 to regulate Pink1 trafficking (Q24297960) (← links)
- Rac1 Protein Rescues Neurite Retraction Caused by G2019S Leucine-rich Repeat Kinase 2 (LRRK2) (Q24298459) (← links)
- Mitofusin 1 and mitofusin 2 are ubiquitinated in a PINK1/parkin-dependent manner upon induction of mitophagy (Q24300975) (← links)
- The role of oxidative stress in Parkinson's disease (Q24307946) (← links)
- Parkin is transcriptionally regulated by ATF4: evidence for an interconnection between mitochondrial stress and ER stress (Q24309549) (← links)
- Phosphorylation of parkin by Parkinson disease-linked kinase PINK1 activates parkin E3 ligase function and NF-kappaB signaling (Q24313304) (← links)
- Structural insights into oligomerization and mitochondrial remodelling of dynamin 1-like protein (Q24338467) (← links)
- Genome-wide association study confirms SNPs in SNCA and the MAPT region as common risk factors for Parkinson disease (Q24596763) (← links)
- Mitochondria, calcium and cell death: a deadly triad in neurodegeneration (Q24646020) (← links)
- A mitochondrial protein compendium elucidates complex I disease biology (Q24655079) (← links)
- Parkinson's Disease: The Mitochondria-Iron Link (Q26746899) (← links)
- Neuroprotective and Therapeutic Strategies against Parkinson's Disease: Recent Perspectives (Q26747508) (← links)
- The Role of Autophagy, Mitophagy and Lysosomal Functions in Modulating Bioenergetics and Survival in the Context of Redox and Proteotoxic Damage: Implications for Neurodegenerative Diseases (Q26750137) (← links)
- Neuroprotective Transcription Factors in Animal Models of Parkinson Disease (Q26765693) (← links)
- The glucagon-like peptide 1 (GLP) receptor as a therapeutic target in Parkinson's disease: mechanisms of action (Q26768527) (← links)
- The Role of Oxidative Stress in Neurodegenerative Diseases (Q26771758) (← links)
- Unfolded Protein Response and Macroautophagy in Alzheimer's, Parkinson's and Prion Diseases (Q26773443) (← links)
- Redox control of protein degradation (Q26783566) (← links)
- Oxidative stress and Parkinson's disease (Q26798897) (← links)
- Parkinson's disease, insulin resistance and novel agents of neuroprotection (Q26853311) (← links)
- Metabolomics of human brain aging and age-related neurodegenerative diseases (Q26992344) (← links)
- Metabolic disturbances in diseases with neurological involvement (Q26999015) (← links)
- Somatic mutations in aging, cancer and neurodegeneration (Q27000745) (← links)
- Upstream deregulation of calcium signaling in Parkinson's disease (Q27026384) (← links)
- Mechanisms of mitophagy (Q27310261) (← links)
- Oxidative stress in Alzheimer's and Parkinson's diseases: insights from the yeast Saccharomyces cerevisiae (Q27692067) (← links)
- Converging roles of ion channels, calcium, metabolic stress, and activity pattern of Substantia nigra dopaminergic neurons in health and Parkinson's disease (Q28074143) (← links)
- The relationship between glucocerebrosidase mutations and Parkinson disease (Q28077257) (← links)
- Effects of 6-hydroxydopamine exposure on motor activity and biochemical expression in zebrafish (Danio rerio) larvae (Q28468677) (← links)
- Parkin is protective against proteotoxic stress in a transgenic zebrafish model (Q28474988) (← links)
- Mutant Parkin impairs mitochondrial function and morphology in human fibroblasts (Q28475596) (← links)
- Bioenergetic consequences of PINK1 mutations in Parkinson disease (Q28477544) (← links)
- Oxidant stress evoked by pacemaking in dopaminergic neurons is attenuated by DJ-1 (Q28510145) (← links)
- Selenoprotein T Exerts an Essential Oxidoreductase Activity That Protects Dopaminergic Neurons in Mouse Models of Parkinson's Disease (Q28854286) (← links)
- PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy (Q29616005) (← links)
- Parkinson's Disease and Cognitive Impairment (Q30239814) (← links)
- Brain mitochondrial dysfunction and oxidative damage in Parkinson's disease. (Q30382570) (← links)
- Relationships among molecular genetic and respiratory properties of Parkinson's disease cybrid cells show similarities to Parkinson's brain tissues (Q30437451) (← links)
- Reduced axonal transport in Parkinson's disease cybrid neurites is restored by light therapy (Q30439165) (← links)
- Hyperexcitable substantia nigra dopamine neurons in PINK1- and HtrA2/Omi-deficient mice (Q30497725) (← links)
- Bioenergetics of neurons inhibit the translocation response of Parkin following rapid mitochondrial depolarization. (Q30498087) (← links)
- Calcium entry induces mitochondrial oxidant stress in vagal neurons at risk in Parkinson's disease (Q30525353) (← links)
- Enteric neurons from Parkinson's disease patients display ex vivo aberrations in mitochondrial structure. (Q30810965) (← links)
- Computational deconvolution of genome wide expression data from Parkinson's and Huntington's disease brain tissues using population-specific expression analysis (Q30885974) (← links)
- Genes associated with Parkinson syndrome (Q33346009) (← links)
- Bridging high-throughput genetic and transcriptional data reveals cellular responses to alpha-synuclein toxicity (Q33411281) (← links)