Pages that link to "Q36420890"

The following pages link to Cardiomyocyte-restricted overexpression of extracellular superoxide dismutase increases nitric oxide bioavailability and reduces infarct size after ischemia/reperfusion (Q36420890):

Displaying 19 items.

• Paradoxical Roles of Antioxidant Enzymes: Basic Mechanisms and Health Implications (Q28392715) (← links)
• Ischemia-reperfusion injury in stroke (Q33653045) (← links)
• Deficiency of ataxia telangiectasia mutated kinase modulates cardiac remodeling following myocardial infarction: involvement in fibrosis and apoptosis (Q35073104) (← links)
• Loss of SOD3 (EcSOD) Expression Promotes an Aggressive Phenotype in Human Pancreatic Ductal Adenocarcinoma. (Q35251686) (← links)
• Preconditioning c-Kit-positive Human Cardiac Stem Cells with a Nitric Oxide Donor Enhances Cell Survival through Activation of Survival Signaling Pathways (Q36850078) (← links)
• Genetic Deficiency of Glutathione S-Transferase P Increases Myocardial Sensitivity to Ischemia-Reperfusion Injury. (Q36862431) (← links)
• Loss of Mrp1 Potentiates Doxorubicin-Induced Cytotoxicity in Neonatal Mouse Cardiomyocytes and Cardiac Fibroblasts (Q37023604) (← links)
• Kinetic and mechanistic considerations to assess the biological fate of peroxynitrite (Q37381268) (← links)
• Reoxygenation-derived toxic reactive oxygen/nitrogen species modulate the contribution of bone marrow progenitor cells to remodeling after myocardial infarction (Q37648491) (← links)
• Extracellular superoxide dismutase is necessary to maintain renal blood flow during sepsis development. (Q37707169) (← links)
• Signaling by S-nitrosylation in the heart (Q38179749) (← links)
• Mitochondrial Function, Biology, and Role in Disease: A Scientific Statement From the American Heart Association. (Q38820532) (← links)
• Cardiac myocyte-specific transgenic ecSOD targets mitochondria to protect against Ca(2+) induced permeability transition (Q42145276) (← links)
• Characterization of oxygen radical formation mechanism at early cardiac ischemia (Q42149344) (← links)
• Low T3 State Is Correlated with Cardiac Mitochondrial Impairments after Ischemia Reperfusion Injury: Evidence from a Proteomic Approach (Q43152861) (← links)
• Exercise-induced cardioprotection: a role for eNOS uncoupling and NO metabolites. (Q45941940) (← links)
• The extracellular isoform of superoxide dismutase has a significant impact on cardiovascular ischaemia and reperfusion injury during cardiopulmonary bypass (Q51247050) (← links)
• Exercise and Cardioprotection: A Natural Defense Against Lethal Myocardial Ischemia-Reperfusion Injury and Potential Guide to Cardiovascular Prophylaxis (Q90447340) (← links)
• Extracellular superoxide dismutase, a molecular transducer of health benefits of exercise (Q90710206) (← links)