Pages that link to "Q36315675"
Jump to navigation
Jump to search
The following pages link to In vivo changes in the patterns of chromatin structure associated with the latent herpes simplex virus type 1 genome in mouse trigeminal ganglia can be detected at early times after butyrate treatment (Q36315675):
Displaying 50 items.
- The role of the CoREST/REST repressor complex in herpes simplex virus 1 productive infection and in latency (Q26823532) (← links)
- A comparison of herpes simplex virus type 1 and varicella-zoster virus latency and reactivation (Q26996418) (← links)
- Ocular Herpes Simplex Virus: How are Latency, Reactivation, Recurrent Disease and Therapy Interrelated? (Q27021069) (← links)
- Neuronal Stress Pathway Mediating a Histone Methyl/Phospho Switch Is Required for Herpes Simplex Virus Reactivation (Q30689513) (← links)
- Role of chromatin during herpesvirus infections (Q33346968) (← links)
- De novo synthesis of VP16 coordinates the exit from HSV latency in vivo (Q33422983) (← links)
- Chromatin at the intersection of viral infection and DNA damage (Q33729186) (← links)
- Control of alpha-herpesvirus IE gene expression by HCF-1 coupled chromatin modification activities (Q33729222) (← links)
- Epigenetic regulation of latent HSV-1 gene expression (Q33729340) (← links)
- Changes to euchromatin on LAT and ICP4 following reactivation are more prevalent in an efficiently reactivating strain of HSV-1. (Q33747905) (← links)
- Disruption of HDAC/CoREST/REST repressor by dnREST reduces genome silencing and increases virulence of herpes simplex virus (Q34115558) (← links)
- Snapshots: chromatin control of viral infection (Q34315920) (← links)
- Upregulation of Mouse Genes in HSV-1 Latent TG after Butyrate Treatment Implicates the Multiple Roles of theLAT-ICP0Locus (Q35005866) (← links)
- The Checkpoints of Viral Gene Expression in Productive and Latent Infection: the Role of the HDAC/CoREST/LSD1/REST Repressor Complex (Q35140220) (← links)
- Characterization of herpes simplex virus 2 primary microRNA Transcript regulation (Q35488636) (← links)
- Inhibition of LSD1 reduces herpesvirus infection, shedding, and recurrence by promoting epigenetic suppression of viral genomes (Q35559725) (← links)
- Dynamic modulation of HSV chromatin drives initiation of infection and provides targets for epigenetic therapies (Q35582225) (← links)
- Decreased reactivation of a herpes simplex virus type 1 (HSV-1) latency-associated transcript (LAT) mutant using the in vivo mouse UV-B model of induced reactivation (Q36199997) (← links)
- Rabbit and mouse models of HSV-1 latency, reactivation, and recurrent eye diseases (Q36307684) (← links)
- CTCF occupation of the herpes simplex virus 1 genome is disrupted at early times postreactivation in a transcription-dependent manner (Q36397366) (← links)
- HSV-1 latent rabbits shed viral DNA into their saliva (Q36459433) (← links)
- A novel selective LSD1/KDM1A inhibitor epigenetically blocks herpes simplex virus lytic replication and reactivation from latency (Q36591414) (← links)
- HSV carrying WT REST establishes latency but reactivates only if the synthesis of REST is suppressed (Q36598134) (← links)
- Targeting the JMJD2 histone demethylases to epigenetically control herpesvirus infection and reactivation from latency (Q36652895) (← links)
- Association of the cellular coactivator HCF-1 with the Golgi apparatus in sensory neurons (Q36898912) (← links)
- The dynamics of HCF-1 modulation of herpes simplex virus chromatin during initiation of infection (Q37015734) (← links)
- Chromatin control of herpes simplex virus lytic and latent infection (Q37080840) (← links)
- Role of polycomb proteins in regulating HSV-1 latency (Q37083714) (← links)
- Recruitment of the transcriptional coactivator HCF-1 to viral immediate-early promoters during initiation of reactivation from latency of herpes simplex virus type 1. (Q37333973) (← links)
- Inhibition of the histone demethylase LSD1 blocks alpha-herpesvirus lytic replication and reactivation from latency (Q37439584) (← links)
- Histone Deacetylase Inhibitors Reduce the Number of Herpes Simplex Virus-1 Genomes Initiating Expression in Individual Cells (Q37474967) (← links)
- Epigenetic repression of herpes simplex virus infection by the nucleosome remodeler CHD3 (Q37519890) (← links)
- Role of viral chromatin structure in the regulation of herpes simplex virus 1 gene expression and replication. (Q37573770) (← links)
- Prior Corneal Scarification and Injection of Immune Serum are Not Required Before Ocular HSV-1 Infection for UV-B-Induced Virus Reactivation and Recurrent Herpetic Corneal Disease in Latently Infected Mice (Q37699104) (← links)
- How to control an infectious bead string: nucleosome-based regulation and targeting of herpesvirus chromatin. (Q37871526) (← links)
- Checkpoints in productive and latent infections with herpes simplex virus 1: conceptualization of the issues (Q37952586) (← links)
- Histone modifications in herpesvirus infections (Q37970405) (← links)
- The 3 facets of regulation of herpes simplex virus gene expression: A critical inquiry (Q38378173) (← links)
- Therapeutics Targeting Protein Acetylation Perturb Latency of Human Viruses (Q38723116) (← links)
- Intrinsic and Innate Defenses of Neurons: Détente with the Herpesviruses (Q38994414) (← links)
- Epigenetic modulation of gene expression from quiescent herpes simplex virus genomes (Q39836698) (← links)
- Transcriptional coactivators are not required for herpes simplex virus type 1 immediate-early gene expression in vitro (Q42043652) (← links)
- Herpes simplex virus 1 DNA is in unstable nucleosomes throughout the lytic infection cycle, and the instability of the nucleosomes is independent of DNA replication (Q42103639) (← links)
- During lytic infections, herpes simplex virus type 1 DNA is in complexes with the properties of unstable nucleosomes (Q42553697) (← links)
- Regulation of histone deposition on the herpes simplex virus type 1 genome during lytic infection (Q43127638) (← links)
- The loss of binary: Pushing the herpesvirus latency paradigm (Q47283029) (← links)
- CTCF binding sites in the HSV-1 genome display site-specific CTCF occupation, protein recruitment and insulator function (Q50043336) (← links)
- Depletion of the insulator protein CTCF results in HSV-1 reactivation in vivo. (Q52668673) (← links)
- Strength in diversity: Understanding the pathways to herpes simplex virus reactivation (Q56333920) (← links)
- Disturbed Yin-Yang balance: stress increases the susceptibility to primary and recurrent infections of herpes simplex virus type 1 (Q90094364) (← links)