Pages that link to "Q35149277"
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The following pages link to NF-kappaB-dependent transcriptional regulation of the cardiac scn5a sodium channel by angiotensin II. (Q35149277):
Displaying 50 items.
- GPD1L links redox state to cardiac excitability by PKC-dependent phosphorylation of the sodium channel SCN5A (Q24323030) (← links)
- Reactive Oxygen Species, Endoplasmic Reticulum Stress and Mitochondrial Dysfunction: The Link with Cardiac Arrhythmogenesis (Q26740623) (← links)
- Electrophysiological Mechanisms of Bayés Syndrome: Insights from Clinical and Mouse Studies (Q26746021) (← links)
- Cellular and Molecular Mechanisms of Arrhythmia by Oxidative Stress (Q26765076) (← links)
- Angiotensin II-superoxide-NFκB signaling and aortic baroreceptor dysfunction in chronic heart failure (Q26777284) (← links)
- Mechanisms of sudden cardiac death: oxidants and metabolism (Q27024176) (← links)
- TGF-β1, released by myofibroblasts, differentially regulates transcription and function of sodium and potassium channels in adult rat ventricular myocytes (Q28573894) (← links)
- Elevated nuclear Foxo1 suppresses excitability of skeletal muscle fibers (Q30544004) (← links)
- Redox regulation, NF-kappaB, and atrial fibrillation. (Q33644314) (← links)
- Mitochondria and arrhythmias. (Q33896746) (← links)
- Deletion of FoxO1 leads to shortening of QRS by increasing Na(+) channel activity through enhanced expression of both cardiac NaV1.5 and β3 subunit (Q34154347) (← links)
- Reactive oxygen species suppress cardiac NaV1.5 expression through Foxo1. (Q34188064) (← links)
- Reduced expression and activation of voltage‐gated sodium channels contributes to blunted baroreflex sensitivity in heart failure rats (Q34192179) (← links)
- Effective antioxidant therapy for the management of arrhythmia (Q34400181) (← links)
- Redox signaling in cardiac myocytes. (Q34629326) (← links)
- Role of inflammation in atrial fibrillation pathophysiology and management. (Q35688023) (← links)
- Metabolic stress, reactive oxygen species, and arrhythmia. (Q35690954) (← links)
- Angiotensin II Induced Cardiac Dysfunction on a Chip (Q35903561) (← links)
- Mitochondria-derived superoxide and voltage-gated sodium channels in baroreceptor neurons from chronic heart-failure rats (Q35951204) (← links)
- Reactive oxygen species-targeted therapeutic interventions for atrial fibrillation. (Q36193817) (← links)
- RBM25/LUC7L3 function in cardiac sodium channel splicing regulation of human heart failure. (Q36494631) (← links)
- Cardiac sodium channel mutations: why so many phenotypes? (Q36932045) (← links)
- The renin-angiotensin-aldosterone system (RAAS) and cardiac arrhythmias (Q37011175) (← links)
- Angiotensin II and oxidative stress in the failing heart (Q37166953) (← links)
- NADPH oxidases and angiotensin II receptor signaling (Q37342232) (← links)
- Genetic variation in angiotensin-converting enzyme-related pathways associated with sudden cardiac arrest risk (Q37375407) (← links)
- NADPH oxidase signaling and cardiac myocyte function. (Q37449222) (← links)
- In vivo transfection of manganese superoxide dismutase gene or nuclear factor κB shRNA in nodose ganglia improves aortic baroreceptor function in heart failure rats (Q37483401) (← links)
- Effect of Shensong Yangxin on the Progression of Paroxysmal Atrial Fibrillation is Correlated with Regulation of Autonomic Nerve Activity (Q37615562) (← links)
- Probucol prevents atrial ion channel remodeling in an alloxan-induced diabetes rabbit model (Q37708905) (← links)
- Biology of cardiac sodium channel Nav1.5 expression (Q37936683) (← links)
- Do multiple nuclear factor kappa B activation mechanisms explain its varied effects in the heart? (Q38093421) (← links)
- A review of mixing and propulsion of chyme in the small intestine: fresh insights from new methods. (Q38343039) (← links)
- Regulation of the cardiac Na+ channel NaV1.5 by post-translational modifications. (Q38369684) (← links)
- The cardiac sodium channel gene SCN5A and its gene product NaV1.5: Role in physiology and pathophysiology. (Q38585131) (← links)
- Calcium-dependent Nedd4-2 upregulation mediates degradation of the cardiac sodium channel Nav1.5: implications for heart failure (Q38906377) (← links)
- Alogliptin, a Dipeptidyl Peptidase-4 Inhibitor, Alleviates Atrial Remodeling and Improves Mitochondrial Function and Biogenesis in Diabetic Rabbits (Q41104564) (← links)
- Extracellular signal-regulated kinase activation during cardiac hypertrophy reduces sarcoplasmic/endoplasmic reticulum calcium ATPase 2 (SERCA2) transcription (Q42055700) (← links)
- Defining the role of oxidative stress in atrial fibrillation and diabetes (Q42059420) (← links)
- Activation of Wnt/β-catenin signaling by hydrogen peroxide transcriptionally inhibits NaV1.5 expression (Q42158072) (← links)
- Irbesartan prevents sodium channel remodeling in a canine model of atrial fibrillation (Q49600485) (← links)
- HuR-mediated SCN5A mRNA stability reduces arrhythmic risk in heart failure (Q49895325) (← links)
- RNA Binding Protein, HuR, Regulates SCN5A Expression Through Stabilizing MEF2C transcription factor mRNA. (Q55499128) (← links)
- Mitochondrial Dysfunction-Associated Arrhythmogenic Substrates in Diabetes Mellitus (Q60046140) (← links)
- Antioxidant Therapy Reduces Oxidative and Inflammatory Tissue Damage in Patients Subjected to Cardiac Surgery with Extracorporeal Circulation (Q82785730) (← links)
- WITHDRAWN: Inhibition of NADPH oxidase up-regulates connexin 43 and ameliorates electrical remodeling in rabbits with heart failure (Q85175672) (← links)
- Arrhythmogenic mechanisms of obstructive sleep apnea in heart failure patients (Q90282403) (← links)
- Anti-arrhythmic properties of non-antiarrhythmic medications (Q90685517) (← links)
- Xenopus Oocyte's Conductance for Bioactive Compounds Screening and Characterization (Q91613445) (← links)
- A small-molecule LF3 abrogates β-catenin/TCF4-mediated suppression of NaV1.5 expression in HL-1 cardiomyocytes (Q92662040) (← links)