Pages that link to "Q34411887"
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The following pages link to The hexosamine biosynthetic pathway couples growth factor-induced glutamine uptake to glucose metabolism (Q34411887):
Displaying 50 items.
- The evolution of genome-scale models of cancer metabolism (Q21129241) (← links)
- N-Acetylglucosamine Inhibits T-helper 1 (Th1)/T-helper 17 (Th17) Cell Responses and Treats Experimental Autoimmune Encephalomyelitis (Q22248070) (← links)
- Phosphofructokinase 1 glycosylation regulates cell growth and metabolism (Q24297510) (← links)
- Erk regulation of pyruvate dehydrogenase flux through PDK4 modulates cell proliferation (Q24323390) (← links)
- Links between metabolism and cancer (Q24626229) (← links)
- Hijacking the Hexosamine Biosynthetic Pathway to Promote EMT-Mediated Neoplastic Phenotypes (Q26752391) (← links)
- Genetics and biology of pancreatic ductal adenocarcinoma (Q26765438) (← links)
- Energy Metabolism Plays a Critical Role in Stem Cell Maintenance and Differentiation (Q26768456) (← links)
- Glutamine and cancer: cell biology, physiology, and clinical opportunities (Q27011818) (← links)
- The emerging paradigm of network medicine in the study of human disease (Q27026041) (← links)
- Selection of metastatic breast cancer cells based on adaptability of their metabolic state (Q28730146) (← links)
- Metabolic reprogramming: a cancer hallmark even warburg did not anticipate (Q29617612) (← links)
- Glycolysis and glutaminolysis cooperatively control T cell function by limiting metabolite supply to N-glycosylation. (Q30365133) (← links)
- Modification of RelA by O-linked N-acetylglucosamine links glucose metabolism to NF-κB acetylation and transcription (Q30419967) (← links)
- A metabolic perturbation by U0126 identifies a role for glutamine in resveratrol-induced cell death (Q30423585) (← links)
- MUC1 facilitates metabolomic reprogramming in triple-negative breast cancer. (Q33626753) (← links)
- AMPK regulates histone H2B O-GlcNAcylation (Q33635491) (← links)
- N-glycan remodeling on glucagon receptor is an effector of nutrient sensing by the hexosamine biosynthesis pathway (Q33718411) (← links)
- The complex landscape of pancreatic cancer metabolism (Q33827703) (← links)
- From Krebs to clinic: glutamine metabolism to cancer therapy. (Q33834498) (← links)
- Interplay between epigenetics and metabolism in oncogenesis: mechanisms and therapeutic approaches (Q33835913) (← links)
- MPI depletion enhances O-GlcNAcylation of p53 and suppresses the Warburg effect. (Q33866466) (← links)
- Collagen-derived proline promotes pancreatic ductal adenocarcinoma cell survival under nutrient limited conditions. (Q33891283) (← links)
- Akt-dependent metabolic reprogramming regulates tumor cell histone acetylation (Q34117812) (← links)
- Prolonged exposure to a Mer ligand in leukemia: Gas6 favors expression of a partial Mer glycoform and reveals a novel role for Mer in the nucleus (Q34171209) (← links)
- Endothelial cell metabolism: parallels and divergences with cancer cell metabolism (Q34228247) (← links)
- A two-way street: reciprocal regulation of metabolism and signalling. (Q34259242) (← links)
- Increased sugar uptake promotes oncogenesis via EPAC/RAP1 and O-GlcNAc pathways (Q34390494) (← links)
- Metabolic reprogramming and dysregulated metabolism: cause, consequence and/or enabler of environmental carcinogenesis? (Q34482056) (← links)
- Autophagy is essential for effector CD8(+) T cell survival and memory formation (Q34516905) (← links)
- Study of global transcriptional changes of N-GlcNAc2 proteins-producing T24 bladder carcinoma cells under glucose deprivation (Q34656404) (← links)
- The adaptor protein p66Shc inhibits mTOR-dependent anabolic metabolism (Q34663562) (← links)
- Mesenchymal phenotype predisposes lung cancer cells to impaired proliferation and redox stress in response to glutaminase inhibition (Q34691457) (← links)
- Golgi N-glycan branching N-acetylglucosaminyltransferases I, V and VI promote nutrient uptake and metabolism. (Q34771274) (← links)
- The metabolic responses to hepatitis B virus infection shed new light on pathogenesis and targets for treatment (Q35075630) (← links)
- Roles of Glucose in Photoreceptor Survival (Q35266544) (← links)
- Loss of the tumor suppressor Hace1 leads to ROS-dependent glutamine addiction (Q35287869) (← links)
- O-linked β-N-acetylglucosamine transferase directs cell proliferation in idiopathic pulmonary arterial hypertension. (Q35328516) (← links)
- Metabolic circuits in neural stem cells (Q35382648) (← links)
- IKKβ and NF-κB transcription govern lymphoma cell survival through AKT-induced plasma membrane trafficking of GLUT1. (Q35587163) (← links)
- Molecular basis for the differential use of glucose and glutamine in cell proliferation as revealed by synchronized HeLa cells. (Q35641497) (← links)
- Aerobic glycolysis tunes YAP/TAZ transcriptional activity (Q35819661) (← links)
- Critical role of O-Linked β-N-acetylglucosamine transferase in prostate cancer invasion, angiogenesis, and metastasis (Q35879559) (← links)
- Role of Glutamine in Cancer: Therapeutic and Imaging Implications: FIGURE 1 (Q35915917) (← links)
- Cellular metabolism and disease: what do metabolic outliers teach us? (Q35915928) (← links)
- Metabolic Reprogramming by Hexosamine Biosynthetic and Golgi N-Glycan Branching Pathways (Q35955540) (← links)
- Protein Kinase A Activation Promotes Cancer Cell Resistance to Glucose Starvation and Anoikis (Q35957554) (← links)
- The roles of O-linked β-N-acetylglucosamine in cardiovascular physiology and disease (Q35994096) (← links)
- The proline regulatory axis and cancer (Q36050265) (← links)
- Glucose deprivation activates a metabolic and signaling amplification loop leading to cell death (Q36096600) (← links)