Pages that link to "Q28505804"
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The following pages link to Dystonia and cerebellar atrophy in Cacna1a null mice lacking P/Q calcium channel activity (Q28505804):
Displaying 50 items.
- Calcium channel, voltage-dependent, P/Q type, alpha 1A subunit (Q21984376) (← links)
- C-termini of P/Q-type Ca2+ channel alpha1A subunits translocate to nuclei and promote polyglutamine-mediated toxicity (Q24314234) (← links)
- Complete loss of P/Q calcium channel activity caused by a CACNA1A missense mutation carried by patients with episodic ataxia type 2 (Q24536238) (← links)
- Familial hemiplegic migraine mutations increase Ca(2+) influx through single human CaV2.1 channels and decrease maximal CaV2.1 current density in neurons (Q24538702) (← links)
- Effects of familial hemiplegic migraine type 1 mutations on neuronal P/Q-type Ca2+ channel activity and inhibitory synaptic transmission (Q24556684) (← links)
- Ion channels as drug targets in central nervous system disorders (Q27004533) (← links)
- Ablation of Ca(V)2.1 voltage-gated Ca²⁺ channels in mouse forebrain generates multiple cognitive impairments (Q27303054) (← links)
- Modal gating of human CaV2.1 (P/Q-type) calcium channels: I. The slow and the fast gating modes and their modulation by beta subunits (Q27863796) (← links)
- A Single Amino Acid Deletion (ΔF1502) in the S6 Segment of CaV2.1 Domain III Associated with Congenital Ataxia Increases Channel Activity and Promotes Ca2+ Influx (Q28118912) (← links)
- A Cacna1a knockin migraine mouse model with increased susceptibility to cortical spreading depression (Q28248603) (← links)
- Characterization of acute somatosensory pain transmission in P/Q-type Ca(2+) channel mutant mice, leaner (Q28508612) (← links)
- Rab3-interacting molecule gamma isoforms lacking the Rab3-binding domain induce long lasting currents but block neurotransmitter vesicle anchoring in voltage-dependent P/Q-type Ca2+ channels (Q28572143) (← links)
- Decreases in the precision of Purkinje cell pacemaking cause cerebellar dysfunction and ataxia (Q28586378) (← links)
- Paroxysmal dyskinesias in the lethargic mouse mutant (Q28587488) (← links)
- Altered properties of quantal neurotransmitter release at endplates of mice lacking P/Q-type Ca2+ channels (Q28594746) (← links)
- Delayed postnatal loss of P/Q-type calcium channels recapitulates the absence epilepsy, dyskinesia, and ataxia phenotypes of genomic Cacna1a mutations. (Q30499188) (← links)
- T-type channel blockade impairs long-term potentiation at the parallel fiber-Purkinje cell synapse and cerebellar learning (Q30559608) (← links)
- Childhood absence epilepsy: genes, channels, neurons and networks. (Q30835420) (← links)
- Neurobiology of migraine. (Q33186891) (← links)
- Familial hemiplegic migraine (Q33280531) (← links)
- CaV2.1 channelopathies (Q33537218) (← links)
- Animal models of generalized dystonia (Q33844755) (← links)
- Paroxysmal dyskinesias in mice (Q33921453) (← links)
- Bioinformatics analysis of biomarkers and transcriptional factor motifs in Down syndrome (Q34272400) (← links)
- The first knockin mouse model of episodic ataxia type 2 (Q34325294) (← links)
- Interactions between presynaptic Ca2+ channels, cytoplasmic messengers and proteins of the synaptic vesicle release complex. (Q34389025) (← links)
- New ataxic tottering-6j mouse allele containing a Cacna1a gene mutation (Q34406472) (← links)
- Identification of epilepsy genes in human and mouse (Q34432267) (← links)
- Spinocerebellar ataxia type 6: channelopathy or glutamine repeat disorder? (Q34446543) (← links)
- Calcium channels and channelopathies of the central nervous system (Q34560191) (← links)
- Molecular genetics of migraine (Q34981965) (← links)
- Cerebellar ataxia, seizures, premature death, and cardiac abnormalities in mice with targeted disruption of the Cacna2d2 gene. (Q35103107) (← links)
- Splice isoform-specific suppression of the Cav2.1 variant underlying spinocerebellar ataxia type 6. (Q35203559) (← links)
- Patterned Purkinje cell death in the cerebellum (Q35563957) (← links)
- Low-voltage-activated ("T-Type") calcium channels in review (Q35682302) (← links)
- In vivo analysis of voltage-dependent calcium channels (Q35682326) (← links)
- Purkinje cell-specific ablation of Cav2.1 channels is sufficient to cause cerebellar ataxia in mice (Q35851329) (← links)
- Age and gender-dependent alternative splicing of P/Q-type calcium channel EF-hand. (Q35994786) (← links)
- Migraine genetics: an update (Q36133899) (← links)
- Models for generalized seizures. (Q36233186) (← links)
- Stargazer--a mouse to seize! (Q36263033) (← links)
- The functional neuroanatomy of dystonia (Q36340491) (← links)
- Convergent mechanisms in etiologically-diverse dystonias (Q36445234) (← links)
- Molecular pathogenesis of spinocerebellar ataxia type 6. (Q36774733) (← links)
- Evaluation of the 3p21.3 tumour-suppressor gene cluster (Q36833529) (← links)
- Two novel alleles of tottering with distinct Ca(v)2.1 calcium channel neuropathologies (Q37079538) (← links)
- Low-frequency oscillations in the cerebellar cortex of the tottering mouse (Q37086565) (← links)
- The ataxic Cacna1a-mutant mouse rolling nagoya: an overview of neuromorphological and electrophysiological findings (Q37502776) (← links)
- Pain channelopathies (Q37689361) (← links)
- P/Q-type calcium channel modulators (Q38016449) (← links)