Pages that link to "Q24685187"
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The following pages link to Gene expression in Wilms' tumor mimics the earliest committed stage in the metanephric mesenchymal-epithelial transition (Q24685187):
Displaying 50 items.
- The pluripotent renal stem cell regulator SIX2 is activated in renal neoplasms and influences cellular proliferation and migration (Q24299112) (← links)
- Wnt5a signaling induces proliferation and survival of endothelial cells in vitro and expression of MMP-1 and Tie-2 (Q24306687) (← links)
- CITED1 expression in Wilms' tumor and embryonic kidney (Q24337902) (← links)
- STAT1 activation regulates proliferation and differentiation of renal progenitors (Q24338667) (← links)
- The Six1 homeoprotein stimulates tumorigenesis by reactivation of cyclin A1 (Q24564859) (← links)
- Tissue microarray validation of epidermal growth factor receptor and SALL2 in synovial sarcoma with comparison to tumors of similar histology (Q24685075) (← links)
- Frizzled7 as an emerging target for cancer therapy (Q26849278) (← links)
- Two-way communication between the metabolic and cell cycle machineries: the molecular basis (Q27013594) (← links)
- Distinct roles and regulations for HoxD genes in metanephric kidney development (Q27314653) (← links)
- Structure-function analyses of the human SIX1–EYA2 complex reveal insights into metastasis and BOR syndrome (Q27676610) (← links)
- Development and differentiation of the ureteric bud into the ureter in the absence of a kidney collecting system (Q28507290) (← links)
- The WTX tumor suppressor regulates mesenchymal progenitor cell fate specification (Q28587086) (← links)
- Allosteric inhibitors of the Eya2 phosphatase are selective and inhibit Eya2-mediated cell migration (Q30409822) (← links)
- Molecular characterization of Wilms' tumor from a resource-constrained region of sub-Saharan Africa (Q30449310) (← links)
- Race disparities in Wilms tumor incidence and biology. (Q30464239) (← links)
- Deducing the stage of origin of Wilms' tumours from a developmental series of Wt1-mutant mice (Q30660201) (← links)
- Cell type-specific over-expression of chromosome 21 genes in fibroblasts and fetal hearts with trisomy 21. (Q33236637) (← links)
- Characterization of the Six1 homeobox gene in normal mammary gland morphogenesis (Q33523907) (← links)
- Six1 expression is associated with a poor prognosis in patients with glioma (Q33597712) (← links)
- The Dachshund gene in development and hormone-responsive tumorigenesis (Q33637720) (← links)
- Increased Six1 expression is associated with poor prognosis in patients with osteosarcoma (Q33684857) (← links)
- T-cell-based therapies for malignancy and infection in childhood (Q33750862) (← links)
- Comparative proteomic analysis of differentially expressed proteins in primary retinoblastoma tumors (Q33764488) (← links)
- Recurrent somatic mutation in DROSHA induces microRNA profile changes in Wilms tumour (Q33772549) (← links)
- The Drosophila Wilms׳ Tumor 1-Associating Protein (WTAP) homolog is required for eye development (Q33776337) (← links)
- Attenuation of Forkhead signaling by the retinal determination factor DACH1. (Q33859716) (← links)
- Large-scale analysis of expression signatures reveals hidden links among diverse cellular processes (Q33914495) (← links)
- The Eyes Absent phosphatase-transactivator proteins promote proliferation, transformation, migration, and invasion of tumor cells (Q33940667) (← links)
- Wilms' tumor blastemal stem cells dedifferentiate to propagate the tumor bulk (Q33949184) (← links)
- Wilms tumor chromatin profiles highlight stem cell properties and a renal developmental network (Q33961864) (← links)
- Temporal blastemal cell gene expression analysis in the kidney reveals new Wnt and related signaling pathway genes to be essential for Wilms' tumor onset (Q34065557) (← links)
- MicroRNA-185 suppresses tumor growth and progression by targeting the Six1 oncogene in human cancers. (Q34124451) (← links)
- Mesenchymal-to-epithelial transition of intercalating cells in Drosophila renal tubules depends on polarity cues from epithelial neighbours (Q34239073) (← links)
- Deletions of 16q in Wilms tumors localize to blastemal-anaplastic cells and are associated with reduced expression of the IRXB renal tubulogenesis gene cluster. (Q34254608) (← links)
- EWS/FLI1 regulates EYA3 in Ewing sarcoma via modulation of miRNA-708, resulting in increased cell survival and chemoresistance. (Q34283431) (← links)
- MiR-199a regulates cell proliferation and survival by targeting FZD7. (Q34338509) (← links)
- Wt1 ablation and Igf2 upregulation in mice result in Wilms tumors with elevated ERK1/2 phosphorylation (Q34428960) (← links)
- Six1 promotes proliferation of pancreatic cancer cells via upregulation of cyclin D1 expression (Q34634833) (← links)
- Selective methylation of CpGs at regulatory binding sites controls NNAT expression in Wilms tumors (Q34795049) (← links)
- Deregulated homeobox gene expression in cancer: cause or consequence? (Q34932292) (← links)
- Wild type p53 transcriptionally represses the SALL2 transcription factor under genotoxic stress (Q34989369) (← links)
- Loss of heterozygosity at 2q37 in sporadic Wilms' tumor: putative role for miR-562. (Q35005638) (← links)
- SIX2 Effects on Wilms Tumor Biology (Q35028775) (← links)
- CTNNB1 mutations and overexpression of Wnt/beta-catenin target genes in WT1-mutant Wilms' tumors (Q35103587) (← links)
- The SIX1-EYA transcriptional complex as a therapeutic target in cancer (Q35107900) (← links)
- Genetic and Epigenetic Mosaicism in Cancer Precursor Tissues (Q35118470) (← links)
- Immortalization of mouse germ line stem cells. (Q35151330) (← links)
- Comparative methylome analysis identifies new tumour subtypes and biomarkers for transformation of nephrogenic rests into Wilms tumour (Q35163343) (← links)
- Eya2 is required to mediate the pro-metastatic functions of Six1 via the induction of TGF-β signaling, epithelial–mesenchymal transition, and cancer stem cell properties (Q35243673) (← links)
- Drosophila eyes absent is a novel mRNA target of the tristetraprolin (TTP) protein DTIS11 (Q35926324) (← links)