Pages that link to "Q77655748"
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The following pages link to Fibroblasts cultured from distal lower extremities in patients with venous reflux display cellular characteristics of senescence (Q77655748):
Displaying 14 items.
- Stem cell therapy for lower extremity diabetic ulcers: where do we stand? (Q26824484) (← links)
- UVB-induced premature senescence of human diploid skin fibroblasts (Q28219260) (← links)
- A new in vitro model of venous hypertension: the effect of pressure on dermal fibroblasts (Q33430533) (← links)
- Treatment of life-threatening wounds with a combination of allogenic platelet-rich plasma, fibrin glue and collagen matrix, and a literature review (Q33837478) (← links)
- Approach of evolutionary theories of ageing, stress, senescence-like phenotypes, calorie restriction and hormesis from the view point of far-from-equilibrium thermodynamics (Q34667141) (← links)
- From the Hayflick mosaic to the mosaics of ageing. Role of stress-induced premature senescence in human ageing (Q34801705) (← links)
- All-trans-retinoic acid suppresses matrix metalloproteinase activity and increases collagen synthesis in diabetic human skin in organ culture (Q35102907) (← links)
- Deconstructing fibrosis research: do pro-fibrotic signals point the way for chronic dermal wound regeneration? (Q37866690) (← links)
- Reactive oxygen species and bacterial biofilms in diabetic wound healing (Q38986390) (← links)
- Altered proliferative responses of dermal fibroblasts to TGF-beta1 may contribute to chronic venous stasis ulcer (Q40645415) (← links)
- Dermal tissue fibrosis in patients with chronic venous insufficiency is associated with increased transforming growth factor-beta1 gene expression and protein production (Q40913440) (← links)
- Reactive Oxygen Species and NOX Enzymes Are Emerging as Key Players in Cutaneous Wound Repair (Q45834311) (← links)
- The effect of passage number on fibroblast cellular senescence in patients with chronic venous insufficiency with and without ulcer (Q72991884) (← links)
- The proliferative capacity of neonatal skin fibroblasts is reduced after exposure to venous ulcer wound fluid: A potential mechanism for senescence in venous ulcers (Q73066095) (← links)