Pages that link to "Q58329660"

The following pages link to Coenzyme Q10 prevents GDP-sensitive mitochondrial uncoupling, glomerular hyperfiltration and proteinuria in kidneys from db/db mice as a model of type 2 diabetes (Q58329660):

Displaying 40 items.

• Role of ROS and RNS Sources in Physiological and Pathological Conditions (Q26740342) (← links)
• Mitochondria: a new therapeutic target in chronic kidney disease (Q26776397) (← links)
• Activation of hypoxia-inducible factors prevents diabetic nephropathy (Q35027230) (← links)
• Molecular strategies for targeting antioxidants to mitochondria: therapeutic implications. (Q35148462) (← links)
• Progress in pathogenesis of proteinuria (Q36011008) (← links)
• NADPH oxidase inhibition reduces tubular sodium transport and improves kidney oxygenation in diabetes. (Q36042860) (← links)
• Antioxidant treatment attenuates lactate production in diabetic nephropathy (Q36245001) (← links)
• Physical exercise, fasting glucose, and renal hyperfiltration in the general population: the Renal Iohexol Clearance Survey in Tromsø 6 (RENIS-T6). (Q36371075) (← links)
• Effect of coenzyme-q10 on Doxorubicin-induced nephrotoxicity in rats (Q36499369) (← links)
• Glutaredoxin mediated redox effects of coenzyme Q10 treatment in type 1 and type 2 diabetes patients (Q36534861) (← links)
• Coenzyme Q10 prevents peripheral neuropathy and attenuates neuron loss in the db-/db- mouse, a type 2 diabetes model (Q36535440) (← links)
• Impact of the hypoxia-inducible factor-1 α (HIF1A) Pro582Ser polymorphism on diabetes nephropathy (Q36560367) (← links)
• Determinants of kidney oxygen consumption and their relationship to tissue oxygen tension in diabetes and hypertension. (Q37633937) (← links)
• Mitochondrial dysfunction and mitophagy: the beginning and end to diabetic nephropathy? (Q37686261) (← links)
• Perspectives and potential applications of mitochondria-targeted antioxidants in cardiometabolic diseases and type 2 diabetes (Q38104693) (← links)
• Impaired hypoxia-inducible factor (HIF) regulation by hyperglycemia. (Q38229803) (← links)
• Angiotensin II reduces transport-dependent oxygen consumption but increases transport-independent oxygen consumption in immortalized mouse proximal tubular cells (Q39004503) (← links)
• Natural antioxidants in the treatment and prevention of diabetic nephropathy; a potential approach that warrants clinical trials. (Q39169974) (← links)
• Mitochondrial Reactive Oxygen Species and Kidney Hypoxia in the Development of Diabetic Nephropathy (Q39262741) (← links)
• Vascular complications in diabetes: old messages, new thoughts (Q39446720) (← links)
• ADCK4 "reenergizes" nephrotic syndrome (Q42004927) (← links)
• The metabolic syndrome induces early changes in the swine renal medullary mitochondria (Q46392965) (← links)
• Hypoxia-inducible factor-1α activation improves renal oxygenation and mitochondrial function in early chronic kidney disease (Q46744839) (← links)
• Differences in susceptibility to develop parameters of diabetic nephropathy in four mouse strains with type 1 diabetes. (Q46920151) (← links)
• Targeted mitochondrial therapy using MitoQ shows equivalent renoprotection to angiotensin converting enzyme inhibition but no combined synergy in diabetes. (Q47103818) (← links)
• Renal transplantation induces mitochondrial uncoupling, increased kidney oxygen consumption, and decreased kidney oxygen tension (Q48264952) (← links)
• Activation of Nrf2 signaling by natural products-can it alleviate diabetes? (Q49385295) (← links)
• Comparison of acute kidney injury of different aetiology reveals in-common mechanisms of tissue damage (Q50003562) (← links)
• Effects of statins on the kidneys in patients with type 2 diabetes (Q51319553) (← links)
• CoQ10-loaded liposomes combined with UTMD prevented early nephropathy of diabetic rats (Q51731037) (← links)
• Endothelin type A receptor inhibition normalises intrarenal hypoxia in rats used as a model of type 1 diabetes by improving oxygen delivery. (Q52931174) (← links)
• Adenosine A2 a receptor stimulation prevents proteinuria in diabetic rats by promoting an anti-inflammatory phenotype without affecting oxidative stress. (Q52950232) (← links)
• Zinc Attenuates Tubulointerstitial Fibrosis in Diabetic Nephropathy Via Inhibition of HIF Through PI-3K Signaling. (Q53153595) (← links)
• Oxidative Stress, Apoptosis, and Mitochondrial Function in Diabetic Nephropathy. (Q54977897) (← links)
• Metformin attenuates renal medullary hypoxia in diabetic nephropathy through uncoupling protein-2 inhibition (Q57806444) (← links)
• Mitochondrial uncoupling has no effect on microvascular complications in type 2 diabetes (Q61443044) (← links)
• miRNA-214 suppresses oxidative stress in diabetic nephropathy via the ROS/Akt/mTOR signaling pathway and uncoupling protein 2 (Q64120570) (← links)
• Protective effect of berberine on high glucose and hypoxia-induced apoptosis via the modulation of HIF-1α in renal tubular epithelial cells (Q64261107) (← links)
• Reduced adenosine A2a receptor-mediated efferent arteriolar vasodilation contributes to diabetes-induced glomerular hyperfiltration (Q88102812) (← links)
• Effects of coenzyme Q10 intervention on diabetic kidney disease: A systematic review and meta-analysis (Q92708567) (← links)