Pages that link to "Q50962265"
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The following pages link to Novel mouse models of oculopharyngeal muscular dystrophy (OPMD) reveal early onset mitochondrial defects and suggest loss of PABPN1 may contribute to pathology. (Q50962265):
Displaying 9 items.
- Nuclear poly(A) binding protein 1 (PABPN1) and Matrin3 interact in muscle cells and regulate RNA processing (Q47700020) (← links)
- Post-transcriptional regulation of Pabpn1 by the RNA binding protein HuR (Q57160226) (← links)
- Deacetylation Inhibition Reverses PABPN1-Dependent Muscle Wasting (Q64236077) (← links)
- Post-transcriptional regulation of gene expression and human disease (Q88004256) (← links)
- Mouse models for muscular dystrophies: an overview (Q90733720) (← links)
- Inhibition of myostatin improves muscle atrophy in oculopharyngeal muscular dystrophy (OPMD) (Q91843569) (← links)
- Proteomic analysis reveals that wildtype and alanine-expanded nuclear poly(A)-binding protein exhibit differential interactions in skeletal muscle (Q92147790) (← links)
- Mitochondrial localization of PABPN1 in oculopharyngeal muscular dystrophy (Q92512425) (← links)
- Cytoskeletal disorganization underlies PABPN1-mediated myogenic disability (Q100737078) (← links)