Pages that link to "Q46618128"
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The following pages link to Lamination of the cerebral cortex is disturbed in Gli3 mutant mice. (Q46618128):
Displaying 14 items.
- Gli3Is Required for Maintenance and Fate Specification of Cortical Progenitors (Q28507310) (← links)
- The Gli3 hypomorphic mutation Pdn causes selective impairment in the growth, patterning, and axon guidance capability of the lateral ganglionic eminence (Q28513168) (← links)
- The doublesex homolog Dmrt5 is required for the development of the caudomedial cerebral cortex in mammals (Q28591918) (← links)
- A crucial role for primary cilia in cortical morphogenesis (Q28594044) (← links)
- Role of Fgf8 signalling in the specification of rostral Cajal-Retzius cells (Q33561649) (← links)
- A focused in situ hybridization screen identifies candidate transcriptional regulators of thymic epithelial cell development and function (Q34075452) (← links)
- Homeobox genes in obsessive-compulsive disorder (Q35647889) (← links)
- Unraveling the disease pathogenesis behind lethal hydrolethalus syndrome revealed multiple changes in molecular and cellular level (Q37202957) (← links)
- Involvement of astrocyte and oligodendrocyte gene sets in migraine (Q40232474) (← links)
- Suppressor of fused controls cerebellar neuronal differentiation in a manner modulated by GLI3 repressor and Fgf15. (Q48352876) (← links)
- Gli3 controls subplate formation and growth of cortical axons. (Q48405243) (← links)
- Lateral Thalamic Eminence: A Novel Origin for mGluR1/Lot Cells (Q48737397) (← links)
- Direct Interactions Between Gli3, Wnt8b, and Fgfs Underlie Patterning of the Dorsal Telencephalon. (Q50545017) (← links)
- Human evolved regulatory elements modulate genes involved in cortical expansion and neurodevelopmental disease susceptibility (Q92501734) (← links)