Pages that link to "Q45304077"

The following pages link to Phenotypic abnormalities in the YAC128 mouse model of Huntington disease are penetrant on multiple genetic backgrounds and modulated by strain (Q45304077):

Displaying 46 items.

• Neuroprotective effects of inositol 1,4,5-trisphosphate receptor C-terminal fragment in a Huntington's disease mouse model (Q24647950) (← links)
• Mouse models of polyglutamine diseases: review and data table. Part I (Q26853025) (← links)
• AAV-dominant negative tumor necrosis factor (DN-TNF) gene transfer to the striatum does not rescue medium spiny neurons in the YAC128 mouse model of Huntington's disease (Q27334472) (← links)
• Effects of deletion of mutant huntingtin in steroidogenic factor 1 neurons on the psychiatric and metabolic phenotype in the BACHD mouse model of Huntington disease (Q27346849) (← links)
• Characterisation of a C1qtnf5 Ser163Arg knock-in mouse model of late-onset retinal macular degeneration (Q28478012) (← links)
• Longitudinal evaluation of the Hdh(CAG)150 knock-in murine model of Huntington's disease (Q28587809) (← links)
• Onset and progression of behavioral and molecular phenotypes in a novel congenic R6/2 line exhibiting intergenerational CAG repeat stability (Q28741746) (← links)
• Extensive early motor and non-motor behavioral deficits are followed by striatal neuronal loss in knock-in Huntington's disease mice (Q30437914) (← links)
• Force-plate quantification of progressive behavioral deficits in the R6/2 mouse model of Huntington's disease (Q30452232) (← links)
• Genetic background modulates behavioral impairments in R6/2 mice and suggests a role for dominant genetic modifiers in Huntington’s disease pathogenesis (Q30467288) (← links)
• Systematic behavioral evaluation of Huntington's disease transgenic and knock-in mouse models (Q30483013) (← links)
• Premature death and neurologic abnormalities in transgenic mice expressing a mutant huntingtin exon-2 fragment. (Q30499096) (← links)
• Tests to assess motor phenotype in mice: a user's guide (Q33464903) (← links)
• BDNF overexpression in the forebrain rescues Huntington's disease phenotypes in YAC128 mice (Q34338809) (← links)
• Natural genetic variation determines susceptibility to aggregation or toxicity in a C. elegans model for polyglutamine disease (Q35002352) (← links)
• Mismatch repair genes Mlh1 and Mlh3 modify CAG instability in Huntington's disease mice: genome-wide and candidate approaches (Q35034398) (← links)
• Altered Balance of Activity in the Striatal Direct and Indirect Pathways in Mouse Models of Huntington's Disease (Q35055022) (← links)
• Combined Treatment with the Mood Stabilizers Lithium and Valproate Produces Multiple Beneficial Effects in Transgenic Mouse Models of Huntington's Disease (Q35356604) (← links)
• Regional vulnerability in Huntington's disease: fMRI-guided molecular analysis in patients and a mouse model of disease (Q35619936) (← links)
• Mutant huntingtin, abnormal mitochondrial dynamics, defective axonal transport of mitochondria, and selective synaptic degeneration in Huntington's disease (Q35645271) (← links)
• Dopaminergic signaling and striatal neurodegeneration in Huntington's disease (Q35839434) (← links)
• Protein misfolding detected early in pathogenesis of transgenic mouse model of Huntington disease using amyloid seeding assay (Q35880053) (← links)
• Metabolic and behavioral effects of mutant huntingtin deletion in Sim1 neurons in the BACHD mouse model of Huntington's disease (Q36059260) (← links)
• Novel BAC Mouse Model of Huntington's Disease with 225 CAG Repeats Exhibits an Early Widespread and Stable Degenerative Phenotype. (Q36315067) (← links)
• Protection by dietary restriction in the YAC128 mouse model of Huntington's disease: Relation to genes regulating histone acetylation and HTT. (Q36763900) (← links)
• Age-Dependent Resistance to Excitotoxicity in Htt CAG140 Mice and the Effect of Strain Background. (Q36984277) (← links)
• Neocortical expression of mutant huntingtin is not required for alterations in striatal gene expression or motor dysfunction in a transgenic mouse (Q37294750) (← links)
• Mitochondrial structural and functional dynamics in Huntington's disease (Q37356257) (← links)
• Suppressing aberrant GluN3A expression rescues synaptic and behavioral impairments in Huntington's disease models (Q37607024) (← links)
• Genetic modifiers of neurological disease (Q37830879) (← links)
• An in vitro perspective on the molecular mechanisms underlying mutant huntingtin protein toxicity (Q38038981) (← links)
• Choosing an animal model for the study of Huntington's disease (Q38139868) (← links)
• Animal models of Huntington's disease for translation to the clinic: best practices (Q38248687) (← links)
• Silencing mutant huntingtin by adeno-associated virus-mediated RNA interference ameliorates disease manifestations in the YAC128 mouse model of Huntington's disease (Q39795092) (← links)
• An enhanced Q175 knock-in mouse model of Huntington disease with higher mutant huntingtin levels and accelerated disease phenotypes (Q40848200) (← links)
• Partial ablation of mu-opioid receptor rich striosomes produces deficits on a motor-skill learning task (Q41816252) (← links)
• Reduced motivation in the BACHD rat model of Huntington disease is dependent on the choice of food deprivation strategy (Q41979425) (← links)
• Huntington's disease: the case for genetic modifiers (Q42181050) (← links)
• Phosphorylation of huntingtin at Ser421 in YAC128 neurons is associated with protection of YAC128 neurons from NMDA-mediated excitotoxicity and is modulated by PP1 and PP2A. (Q42845671) (← links)
• Striatal expression of a calmodulin fragment improved motor function, weight loss, and neuropathology in the R6/2 mouse model of Huntington's disease. (Q43275673) (← links)
• IGF-1 intranasal administration rescues Huntington's disease phenotypes in YAC128 mice. (Q45296557) (← links)
• Transgenic Animal Models of Huntington’s Disease (Q45301195) (← links)
• Mitochondrial Metabolism in a Large-Animal Model of Huntington Disease: The Hunt for Biomarkers in the Spermatozoa of Presymptomatic Minipigs. (Q45302844) (← links)
• HACE1 is essential for astrocyte mitochondrial function and influences Huntington disease phenotypes in vivo. (Q45305356) (← links)
• Unexpected cell type-dependent effects of autophagy on polyglutamine aggregation revealed by natural genetic variation in C. elegans (Q89863572) (← links)
• Deficit in Motor Skill Consolidation-Dependent Synaptic Plasticity at Motor Cortex to Dorsolateral Striatum Synapses in a Mouse Model of Huntington's Disease (Q90117097) (← links)