Pages that link to "Q42188947"

The following pages link to Role of mononuclear phagocytes in elimination of Plasmodium chabaudi AS infection (Q42188947):

Displaying 28 items.

• CD4+ and CD8+ T lymphocytes both contribute to acquired immunity to blood-stage Plasmodium chabaudi AS (Q24645453) (← links)
• Splenic red pulp macrophages produce type I interferons as early sentinels of malaria infection but are dispensable for control (Q27312553) (← links)
• Altered immune responses in mice with concomitant Schistosoma mansoni and Plasmodium chabaudi infections (Q33767382) (← links)
• Gamma interferon production is critical for protective immunity to infection with blood-stage Plasmodium berghei XAT but neither NO production nor NK cell activation is critical (Q33867123) (← links)
• The immunology of malaria infection (Q33970972) (← links)
• Cellular changes and apoptosis in the spleens and peripheral blood of mice infected with blood-stage Plasmodium chabaudi chabaudi AS. (Q34003620) (← links)
• Resistance to malaria by enhanced phagocytosis of erythrocytes in LMP7-deficient mice (Q34635361) (← links)
• Complex genetic control of susceptibility to malaria in mice (Q34679667) (← links)
• Macrophages are the determinant of resistance to and outcome of nonlethal Babesia microti infection in mice (Q34890110) (← links)
• Investigating the evolution of apoptosis in malaria parasites: the importance of ecology (Q35104718) (← links)
• Role of endogenous gamma interferon in host response to infection with blood-stage Plasmodium chabaudi AS. (Q35105662) (← links)
• Macrophages are critical for cross-protective immunity conferred by Babesia microti against Babesia rodhaini infection in mice (Q35665478) (← links)
• Macrophage Colony Stimulating Factor Derived from CD4+ T Cells Contributes to Control of a Blood-Borne Infection (Q36214299) (← links)
• Macrophage-mediated but gamma interferon-independent innate immune responses control the primary wave of Plasmodium yoelii parasitemia (Q36314029) (← links)
• Osteoclasts Are Required for Hematopoietic Stem and Progenitor Cell Mobilization but Not for Stress Erythropoiesis in Plasmodium chabaudi adami Murine Malaria (Q36556173) (← links)
• Depletion of Phagocytic Cells during Nonlethal Plasmodium yoelii Infection Causes Severe Malaria Characterized by Acute Renal Failure in Mice (Q36631393) (← links)
• Macrophage activation during Plasmodium chabaudi AS infection in resistant C57BL/6 and susceptible A/J mice (Q36956663) (← links)
• Protective immunity against malaria after vaccination (Q38159714) (← links)
• Rosiglitazone modulates the innate immune response to Plasmodium falciparum infection and improves outcome in experimental cerebral malaria (Q39446904) (← links)
• Blood transfusion alters the course and outcome of Plasmodium chabaudi AS infection in mice. (Q40375196) (← links)
• The roles of cytokines produced in the immune response to the erythrocytic stages of mouse malarias (Q40724094) (← links)
• The within-host cellular dynamics of bloodstage malaria: theoretical and experimental studies (Q45080742) (← links)
• Early IL-12 p70, but not p40, production by splenic macrophages correlates with host resistance to blood-stage Plasmodium chabaudi AS malaria (Q47851280) (← links)
• The inhibition of Plasmodium falciparum growth in vitro by sera from mice infected with malaria or treated with TNF. (Q47877128) (← links)
• Hepatic Kupffer cell phagocytotic function in rats with erythrocytic-stage malaria (Q47907323) (← links)
• Anaemia and resistance to malaria in transgenic mice expressing human tumour necrosis factor. (Q48010428) (← links)
• Dimorphic Plasmodium falciparum merozoite surface protein-1 epitopes turn off memory T cells and interfere with T cell priming (Q48027581) (← links)
• Innate Immunity to Malaria (Q63952516) (← links)