Pages that link to "Q40474265"

The following pages link to The double-edged sword of activation-induced cytidine deaminase. (Q40474265):

Displaying 43 items.

• Histone chaperone Spt6 is required for class switch recombination but not somatic hypermutation (Q24300395) (← links)
• Adaptive evolution and antiviral activity of the conserved mammalian cytidine deaminase APOBEC3H. (Q24311896) (← links)
• The convergence of DNA damage checkpoint pathways and androgen receptor signaling in prostate cancer (Q27014101) (← links)
• The Biological Effects of IL-21 Signaling on B-Cell-Mediated Responses in Organ Transplantation (Q28078919) (← links)
• First AID (activation-induced cytidine deaminase) is needed to produce high affinity isotype-switched antibodies (Q28235340) (← links)
• FANCI protein binds to DNA and interacts with FANCD2 to recognize branched structures (Q28250124) (← links)
• The interaction between AID and CIB1 is nonessential for antibody gene diversification by gene conversion or class switch recombination (Q28750273) (← links)
• AID and Apobec3G haphazard deamination and mutational diversity (Q30537745) (← links)
• DNA-dependent protein kinase inhibits AID-induced antibody gene conversion (Q33278104) (← links)
• Two forms of activation-induced cytidine deaminase differing in their ability to bind agarose (Q33527420) (← links)
• Regulation of immunoglobulin class-switch recombination: choreography of noncoding transcription, targeted DNA deamination, and long-range DNA repair (Q34115234) (← links)
• Activation-induced deaminase cloning, localization, and protein extraction from young VH-mutant rabbit appendix (Q34130443) (← links)
• The cytidine deaminases AID and APOBEC-1 exhibit distinct functional properties in a novel yeast selectable system (Q34195827) (← links)
• Affinity maturation of anti-TNF-alpha scFv with somatic hypermutation in non-B cells (Q34216459) (← links)
• Regulation of activation-induced deaminase stability and antibody gene diversification by Hsp90. (Q34340626) (← links)
• Epigenetic tethering of AID to the donor switch region during immunoglobulin class switch recombination. (Q35144590) (← links)
• Activation-induced cytidine deaminase (AID)-dependent somatic hypermutation requires a splice isoform of the serine/arginine-rich (SR) protein SRSF1 (Q35709088) (← links)
• Individual substitution mutations in the AID C terminus that ablate IgH class switch recombination (Q35744148) (← links)
• AID to overcome the limitations of genomic information (Q36170796) (← links)
• A structural basis for the biochemical behavior of activation-induced deoxycytidine deaminase class-switch recombination-defective hyper-IgM-2 mutants (Q36201805) (← links)
• Activation-induced cytidine deaminase: structure-function relationship as based on the study of mutants (Q36590567) (← links)
• Antibody diversification: mutational mechanisms and oncogenesis (Q36604773) (← links)
• Discovery of activation-induced cytidine deaminase, the engraver of antibody memory (Q36845653) (← links)
• Pathophysiology of B-cell intrinsic immunoglobulin class switch recombination deficiencies (Q36845690) (← links)
• GANP regulates recruitment of AID to immunoglobulin variable regions by modulating transcription and nucleosome occupancy (Q36906733) (← links)
• Alternative splicing regulates activation-induced cytidine deaminase (AID): implications for suppression of AID mutagenic activity in normal and malignant B cells (Q37001985) (← links)
• Immunoglobulin class switch recombination: study through human natural mutants (Q37140490) (← links)
• A role for host activation-induced cytidine deaminase in innate immune defense against KSHV. (Q37288983) (← links)
• Activation induced deaminase C-terminal domain links DNA breaks to end protection and repair during class switch recombination (Q37659903) (← links)
• Activation-induced cytidine deaminase structure and functions: A species comparative view (Q37846899) (← links)
• Molecular Mechanism of Immunoglobulin V‐Region Diversification Regulated by Transcription and RNA Metabolism in Antigen‐Driven B Cells (Q37851179) (← links)
• The AID dilemma: infection, or cancer? (Q37994927) (← links)
• An evolutionary view of the mechanism for immune and genome diversity (Q38001656) (← links)
• Evolutionary comparison of the mechanism of DNA cleavage with respect to immune diversity and genomic instability (Q38019962) (← links)
• Expression of activation-induced cytidine deaminase is associated with a poor prognosis of diffuse large B cell lymphoma patients treated with CHOP-based chemotherapy. (Q41667047) (← links)
• Active nuclear import and cytoplasmic retention of activation-induced deaminase (Q41792388) (← links)
• AID recruits UNG and Msh2 to Ig switch regions dependent upon the AID C terminus [corrected]. (Q41870415) (← links)
• The mechanisms regulating the subcellular localization of AID (Q41981500) (← links)
• Mismatch repair proteins and AID activity are required for the dominant negative function of C-terminally deleted AID in class switching (Q42837072) (← links)
• Different isoforms of the B-cell mutator activation-induced cytidine deaminase are aberrantly expressed in BCR-ABL1-positive acute lymphoblastic leukemia patients. (Q43275641) (← links)
• Evolution of class switch recombination function in fish activation-induced cytidine deaminase, AID. (Q46807599) (← links)
• A-Miz-ing BCL6. (Q53656452) (← links)
• Activation-induced cytidine deaminase (AID) is localized to subnuclear domains enriched in splicing factors (Q87113173) (← links)