Pages that link to "Q36365206"
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The following pages link to Dimerization of soluble major histocompatibility complex-peptide complexes is sufficient for activation of T cell hybridoma and induction of unresponsiveness (Q36365206):
Displaying 50 items.
- Linking form to function: Biophysical aspects of artificial antigen presenting cell design (Q27002652) (← links)
- Surface-anchored monomeric agonist pMHCs alone trigger TCR with high sensitivity (Q27333772) (← links)
- Phage display of peptide/major histocompatibility complex. (Q30890366) (← links)
- The SCHOOL of nature: I. Transmembrane signaling (Q33351190) (← links)
- Soluble HLA class I molecules: biological significance and clinical implications (Q33368605) (← links)
- The role of T-cell receptor dimerization in T-cell activation. (Q33774239) (← links)
- Soluble peptide-MHC monomers cause activation of CD8+ T cells through transfer of the peptide to T cell MHC molecules (Q34160808) (← links)
- Class I negative CD8 T cells reveal the confounding role of peptide-transfer onto CD8 T cells stimulated with soluble H2-Kb molecules (Q34160870) (← links)
- T-cell activation by soluble MHC oligomers can be described by a two-parameter binding model (Q34176726) (← links)
- Receptor clustering and transmembrane signaling in T cells (Q34245432) (← links)
- Soluble MOG35-55/I-A(b) dimers ameliorate experimental autoimmune encephalomyelitis by reducing encephalitogenic T cells (Q34450848) (← links)
- Generation and use of alternative multimers of peptide/MHC complexes (Q34815646) (← links)
- Multivalent structure of an alphabetaT cell receptor (Q35000179) (← links)
- Tumor antigens and proteomics from the point of view of the major histocompatibility complex peptides (Q35160569) (← links)
- Antigen-specific therapeutic approaches in Type 1 diabetes (Q35762883) (← links)
- In vivo priming of IL-17(+) uveitogenic T cells is enhanced by Toll ligand receptor (TLR)2 and TLR4 agonists via γδ T cell activation (Q35783661) (← links)
- A T cell receptor V alpha domain expressed in bacteria: does it dimerize in solution? (Q36367417) (← links)
- Critical role for CD8 in T cell receptor binding and activation by peptide/major histocompatibility complex multimers (Q36375751) (← links)
- Potent T cell activation with dimeric peptide-major histocompatibility complex class II ligand: the role of CD4 coreceptor (Q36401714) (← links)
- Coexistence of multivalent and monovalent TCRs explains high sensitivity and wide range of response (Q36402779) (← links)
- Superactivation of an immune response triggered by oligomerized T cell epitopes (Q36828919) (← links)
- Covalent assembly of a soluble T cell receptor-peptide-major histocompatibility class I complex (Q37493312) (← links)
- Dimeric MHC-peptides inserted into an immunoglobulin scaffold as new immunotherapeutic agents (Q37857379) (← links)
- Peptide-MHC-based nanovaccines for the treatment of autoimmunity: a "one size fits all" approach? (Q37866131) (← links)
- Receptor Pre-Clustering and T cell Responses: Insights into Molecular Mechanisms (Q38211020) (← links)
- Induction of viral and tumour specific CTL responses using antibody targeted HLA class I peptide complexes (Q38282953) (← links)
- MHC class I dimer formation by alteration of the cellular redox environment and induction of apoptosis (Q39449645) (← links)
- Early T-cell activation biophysics. (Q39654323) (← links)
- Role of class I human leukocyte antigen molecules in early steps of echovirus infection of rhabdomyosarcoma cells. (Q39934665) (← links)
- Study of the Mechanism of TCR Antagonism Using Dual-TCR-Expressing T Cells (Q40653232) (← links)
- The beta1 and beta3 integrins promote T cell receptor-mediated cytotoxic T lymphocyte activation (Q40656302) (← links)
- Receptor proximity, not intermolecular orientation, is critical for triggering T-cell activation (Q40802102) (← links)
- The relationship of MHC-peptide binding and T cell activation probed using chemically defined MHC class II oligomers (Q40831490) (← links)
- Human major histocompatibility molecules have the intrinsic ability to form homotypic associations (Q40877951) (← links)
- Soluble HLA class I molecules induce apoptosis in alloreactive cytotoxic T lymphocytes (Q41172428) (← links)
- T cell antigen receptor ubiquitination is a consequence of receptor-mediated tyrosine kinase activation (Q41206192) (← links)
- Biophysical and structural studies of TCRs and ligands: implications for T cell signaling (Q41353141) (← links)
- Systemic antigen in the treatment of T-cell-mediated autoimmune diseases (Q41679018) (← links)
- Initiation of signal transduction through the T cell receptor requires the multivalent engagement of peptide/MHC ligands [corrected] (Q46842083) (← links)
- Immunologic suppression mediated by genetically modified hepatocytes expressing secreted allo-MHC class I molecules (Q47763415) (← links)
- Formation of TCR dimers/trimers as a crucial step for T cell activation. (Q52237316) (← links)
- A window of opportunity for cooperativity in the T Cell Receptor. (Q55475768) (← links)
- CD8 Expression Allows T Cell Signaling by Monomeric Peptide-MHC Complexes (Q57275526) (← links)
- Induction of T cell anergy by liposomes with incorporated major histocompatibility complex (MHC) II/peptide complexes (Q61651912) (← links)
- Efficient binding of reduced peptide bond pseudopeptides to major histocompatibility complex class I molecule (Q71823807) (← links)
- Split tolerance to the MHC class I molecule H-2Dd in animals transgenic for its soluble analog (Q73170826) (← links)
- Assessment of homozygosity in HLA-class I antigens and their distribution/quantitation in subpopulations of T cells by flow cytometry (Q73279069) (← links)
- Kinetics of T-cell receptor binding by bivalent HLA-DR. Peptide complexes that activate antigen-specific human T-cells (Q73316769) (← links)
- Enrichment of antigen-specific T lymphocytes by panning on immobilized MHC-peptide complexes (Q73886861) (← links)
- A diverse set of oligomeric class II MHC-peptide complexes for probing T-cell receptor interactions (Q74297571) (← links)