Pages that link to "Q36229299"
Jump to navigation
Jump to search
The following pages link to NK cell activation in visceral leishmaniasis requires TLR9, myeloid DCs, and IL-12, but is independent of plasmacytoid DCs. (Q36229299):
Displaying 50 items.
- Possibilities and challenges for developing a successful vaccine for leishmaniasis (Q26749758) (← links)
- Dendritic Cells and Leishmania Infection: Adding Layers of Complexity to a Complex Disease (Q26767147) (← links)
- Innate immunity against Leishmania infections (Q27010187) (← links)
- Innate killing of Leishmania donovani by macrophages of the splenic marginal zone requires IRF-7 (Q27315168) (← links)
- iNOS-producing inflammatory dendritic cells constitute the major infected cell type during the chronic Leishmania major infection phase of C57BL/6 resistant mice (Q27316669) (← links)
- Key role of splenic myeloid DCs in the IFN-alphabeta response to adenoviruses in vivo (Q27318346) (← links)
- Lessons from other diseases: granulomatous inflammation in leishmaniasis (Q27323278) (← links)
- IL-17 mediates immunopathology in the absence of IL-10 following Leishmania major infection (Q27335548) (← links)
- TLR4 and NKT cell synergy in immunotherapy against visceral leishmaniasis (Q27346402) (← links)
- Leishmania interferes with host cell signaling to devise a survival strategy (Q33781265) (← links)
- Critical role of IRF-5 in the development of T helper 1 responses to Leishmania donovani infection (Q33799144) (← links)
- IL-18, but not IL-15, contributes to the IL-12-dependent induction of NK-cell effector functions by Leishmania infantum in vivo. (Q34017578) (← links)
- Leishmania donovani-induced expression of signal regulatory protein alpha on Kupffer cells enhances hepatic invariant NKT-cell activation (Q34017603) (← links)
- Type I IFN receptor regulates neutrophil functions and innate immunity to Leishmania parasites (Q34155021) (← links)
- Glycoinositolphospholipids from Leishmania braziliensis and L. infantum: modulation of innate immune system and variations in carbohydrate structure (Q34184759) (← links)
- Infection-induced regulation of natural killer cells by macrophages and collagen at the lymph node subcapsular sinus (Q34354896) (← links)
- Activation of phosphatidylinositol 3-kinase/Akt and impairment of nuclear factor-kappaB: molecular mechanisms behind the arrested maturation/activation state of Leishmania infantum-infected dendritic cells (Q34358188) (← links)
- Deception and manipulation: the arms of leishmania, a successful parasite (Q34369161) (← links)
- IL-10-producing Th1 cells and disease progression are regulated by distinct CD11c⁺ cell populations during visceral leishmaniasis (Q34387183) (← links)
- TLR9 activation is triggered by the excess of stimulatory versus inhibitory motifs present in Trypanosomatidae DNA. (Q34505652) (← links)
- NK cell activity differs between patients with localized and diffuse cutaneous leishmaniasis infected with Leishmania mexicana: a comparative study of TLRs and cytokines (Q34513470) (← links)
- Mechanisms of resistance and susceptibility to experimental visceral leishmaniosis: BALB/c mouse versus Syrian hamster model. (Q34639315) (← links)
- Expression of Toll-like receptors 2 and 9 in cells of dog jejunum and colon naturally infected with Leishmania infantum. (Q34719048) (← links)
- Leishmania-infected macrophages are targets of NK cell-derived cytokines but not of NK cell cytotoxicity (Q35329183) (← links)
- Antileishmanial effect of 18β-glycyrrhetinic acid is mediated by Toll-like receptor-dependent canonical and noncanonical p38 activation (Q35385952) (← links)
- Cytoskeletal stabilization of inhibitory interactions in immunologic synapses of mature human dendritic cells with natural killer cells (Q35623632) (← links)
- Lymph node hypertrophy following Leishmania major infection is dependent on TLR9. (Q35685422) (← links)
- Cathepsin B-Deficient Mice Resolve Leishmania major Inflammation Faster in a T Cell-Dependent Manner (Q36018448) (← links)
- The role of leishmania proteophosphoglycans in sand fly transmission and infection of the Mammalian host (Q36063617) (← links)
- The early interaction of Leishmania with macrophages and dendritic cells and its influence on the host immune response (Q36159685) (← links)
- NK Cell Proliferation Induced by IL-15 Transpresentation Is Negatively Regulated by Inhibitory Receptors (Q36260218) (← links)
- Toll-like receptor 9 signaling in dendritic cells regulates neutrophil recruitment to inflammatory foci following Leishmania infantum infection. (Q36281302) (← links)
- Toll-like receptors and leishmaniasis. (Q36483521) (← links)
- Leishmania pifanoi proteoglycolipid complex P8 induces macrophage cytokine production through Toll-like receptor 4. (Q36593911) (← links)
- Infection with Vpr-positive human immunodeficiency virus type 1 impairs NK cell function indirectly through cytokine dysregulation of infected target cells. (Q36747731) (← links)
- Role of Toll-like receptor 9 signaling in experimental Leishmania braziliensis infection (Q36827001) (← links)
- Role of natural killer cells in modulating dendritic cell responses to Leishmania amazonensis infection (Q36950033) (← links)
- Down-regulation of dendritic cell signaling pathways by Leishmania amazonensis amastigotes (Q36973139) (← links)
- Evolutionary struggles between NK cells and viruses (Q36976220) (← links)
- Rickettsiae stimulate dendritic cells through toll-like receptor 4, leading to enhanced NK cell activation in vivo (Q37035273) (← links)
- Modulation of dendritic cell function by Leishmania parasites (Q37091427) (← links)
- Dendritic cells are required for optimal activation of natural killer functions following primary infection with herpes simplex virus type 1. (Q37127934) (← links)
- Probing in vivo dendritic cell functions by conditional cell ablation. (Q37139269) (← links)
- Natural killer cells in immunodefense against infective agents (Q37178929) (← links)
- Distinct roles for MyD88 and Toll-like receptor 2 during Leishmania braziliensis infection in mice (Q37256528) (← links)
- Genetic and antibody-mediated reprogramming of natural killer cell missing-self recognition in vivo (Q37293319) (← links)
- Controlling natural killer cell responses: integration of signals for activation and inhibition (Q37404156) (← links)
- TLR9 and MyD88 are crucial for the maturation and activation of dendritic cells by paromomycin-miltefosine combination therapy in visceral leishmaniasis (Q37635270) (← links)
- Modulation of dendritic cell responses by parasites: a common strategy to survive (Q37702795) (← links)
- Mechanisms of NK cell activation: CD4(+) T cells enter the scene. (Q37921196) (← links)