Pages that link to "Q33912964"
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The following pages link to A hierarchical role for classical pathway complement proteins in the clearance of apoptotic cells in vivo (Q33912964):
Displaying 50 items.
- Cytokine-associated neutrophil extracellular traps and antinuclear antibodies in Plasmodium falciparum infected children under six years of age (Q21034151) (← links)
- The Role of the Complement System in Innate Immunity (Q22306048) (← links)
- Specific binding of L-ficolin and H-ficolin to apoptotic cells leads to complement activation (Q24299377) (← links)
- Complement classical pathway components are all important in clearance of apoptotic and secondary necrotic cells (Q24322831) (← links)
- Elastase-mediated phosphatidylserine receptor cleavage impairs apoptotic cell clearance in cystic fibrosis and bronchiectasis (Q24551335) (← links)
- Paths reunited: Initiation of the classical and lectin pathways of complement activation (Q24646047) (← links)
- PPAR-delta senses and orchestrates clearance of apoptotic cells to promote tolerance (Q24650398) (← links)
- Epidermal growth factor-like domain repeat of stabilin-2 recognizes phosphatidylserine during cell corpse clearance (Q24658141) (← links)
- C1q and mannose binding lectin engagement of cell surface calreticulin and CD91 initiates macropinocytosis and uptake of apoptotic cells (Q24670661) (← links)
- ATP-binding cassette transporter A7 enhances phagocytosis of apoptotic cells and associated ERK signaling in macrophages (Q24683025) (← links)
- Phosphatidylserine receptor and apoptosis: consequences of a non-ingested meal (Q24805427) (← links)
- Links between complement deficiency and apoptosis (Q24805502) (← links)
- Genetic epidemiology: systemic lupus erythematosus (Q24805794) (← links)
- Complement in Lupus Nephritis: New Perspectives (Q26740491) (← links)
- Systemic Lupus Erythematosus and Deficiencies of Early Components of the Complement Classical Pathway (Q26768534) (← links)
- Cell death in the pathogenesis of immune-mediated diseases: the role of HMGB1 and DAMP-PAMP complexes (Q27008742) (← links)
- C1q binds phosphatidylserine and likely acts as a multiligand-bridging molecule in apoptotic cell recognition (Q27649785) (← links)
- Identification of a factor that links apoptotic cells to phagocytes (Q28217034) (← links)
- Immune response against dying tumor cells (Q28271233) (← links)
- Neutrophils regulate tissue Neutrophilia in inflammation via the oxidant-modified lipid lysophosphatidylserine (Q28282760) (← links)
- Signaling via macrophage G2A enhances efferocytosis of dying neutrophils by augmentation of Rac activity (Q28304843) (← links)
- A new triggering receptor expressed on myeloid cells (Trem) family member, Trem-like 4, binds to dead cells and is a DNAX activation protein 12-linked marker for subsets of mouse macrophages and dendritic cells (Q28511097) (← links)
- The endothelial deprotection hypothesis for lupus pathogenesis: the dual role of C1q as a mediator of clearance and regulator of endothelial permeability (Q28649846) (← links)
- Resolution of inflammation: the beginning programs the end (Q29615695) (← links)
- The role of nucleotides in apoptotic cell clearance: implications for disease pathogenesis (Q30429149) (← links)
- ACAT inhibition reduces the progression of preexisting, advanced atherosclerotic mouse lesions without plaque or systemic toxicity (Q30538414) (← links)
- Complement receptor 3 (CD11b/CD18) is implicated in the elimination of β-amyloid peptides (Q30665192) (← links)
- Complement in glomerular injury (Q33376840) (← links)
- Complement regulators and inhibitory proteins (Q33386110) (← links)
- C1q differentially modulates phagocytosis and cytokine responses during ingestion of apoptotic cells by human monocytes, macrophages, and dendritic cells. (Q33745559) (← links)
- Complement C4 maintains peripheral B-cell tolerance in a myeloid cell dependent manner. (Q33861070) (← links)
- Isoflurane promotes phagocytosis of apoptotic neutrophils through AMPK-mediated ADAM17/Mer signaling (Q33865863) (← links)
- Endogenous NO upon estradiol-17β stimulation and NO donor differentially regulate mitochondrial S-nitrosylation in endothelial cells (Q33901272) (← links)
- Corpse clearance defines the meaning of cell death (Q33922730) (← links)
- Phagocytosis and clearance of apoptotic cells is mediated by MER. (Q33946282) (← links)
- Plasminogen promotes macrophage phagocytosis in mice (Q33987072) (← links)
- Gene expression profiling of dendritic cells reveals important mechanisms associated with predisposition to Staphylococcus infections (Q33998838) (← links)
- A polymorphism in the complement component C1qA correlates with prolonged response following rituximab therapy of follicular lymphoma (Q34007430) (← links)
- Systemic lupus erythematosus, complement deficiency, and apoptosis (Q34082803) (← links)
- Molecular mechanisms of late apoptotic/necrotic cell clearance (Q34088856) (← links)
- Apoptotic cell-based therapies against transplant rejection: role of recipient's dendritic cells (Q34089049) (← links)
- Wound-healing defect of CD18(-/-) mice due to a decrease in TGF-beta1 and myofibroblast differentiation (Q34116215) (← links)
- The complement cascade as a mediator of tissue growth and regeneration (Q34154941) (← links)
- A blast from the past: clearance of apoptotic cells regulates immune responses. (Q34162195) (← links)
- Native polymeric forms of properdin selectively bind to targets and promote activation of the alternative pathway of complement (Q34172424) (← links)
- Characterization of the effects of cross-linking of macrophage CD44 associated with increased phagocytosis of apoptotic PMN. (Q34200530) (← links)
- Dendritic cells resurrect antigens from dead cells. (Q34207623) (← links)
- Studies of the humoral immune response (Q34242639) (← links)
- Apoptosis, clearance mechanisms, and the development of systemic lupus erythematosus. (Q34250889) (← links)
- Regulation of phagocytosis by TAM receptors and their ligands (Q34271347) (← links)