Pages that link to "Q30479480"
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The following pages link to Slc4a11 gene disruption in mice: cellular targets of sensorineuronal abnormalities (Q30479480):
Displaying 33 items.
- Fuchs endothelial corneal dystrophy: current perspectives (Q26764908) (← links)
- SLC4A11 and the Pathophysiology of Congenital Hereditary Endothelial Dystrophy (Q26785510) (← links)
- SLC4A11 prevents osmotic imbalance leading to corneal endothelial dystrophy, deafness, and polyuria. (Q28590585) (← links)
- Genetics of peripheral vestibular dysfunction: lessons from mutant mouse strains (Q30420179) (← links)
- The divergence, actions, roles, and relatives of sodium-coupled bicarbonate transporters (Q30440948) (← links)
- Immunocytochemical distribution of WARP (von Willebrand A domain-related protein) in the inner ear. (Q30471582) (← links)
- Depletion of SLC4A11 causes cell death by apoptosis in an immortalized human corneal endothelial cell line (Q33556321) (← links)
- Glutaminolysis is Essential for Energy Production and Ion Transport in Human Corneal Endothelium. (Q33809428) (← links)
- Congenital hereditary endothelial dystrophy caused by SLC4A11 mutations progresses to Harboyan syndrome (Q34331533) (← links)
- The SLC4 family of bicarbonate (HCO₃⁻) transporters (Q34333694) (← links)
- Fuchs Endothelial Corneal Dystrophy (Q34624570) (← links)
- Human SLC4A11-C functions as a DIDS-stimulatable H⁺(OH⁻) permeation pathway: partial correction of R109H mutant transport (Q34980472) (← links)
- Molecular bases of corneal endothelial dystrophies (Q35739942) (← links)
- Human SLC4A11 Is a Novel NH3/H+ Co-transporter. (Q35860561) (← links)
- The genetics of Fuchs' corneal dystrophy (Q36754678) (← links)
- SLC4A11 is an EIPA-sensitive Na(+) permeable pHi regulator. (Q37234550) (← links)
- Transmembrane water-flux through SLC4A11: a route defective in genetic corneal diseases. (Q37470604) (← links)
- Genetics of corneal endothelial dystrophies (Q37678805) (← links)
- Loss of DDRGK1 modulates SOX9 ubiquitination in spondyloepimetaphyseal dysplasia (Q37730619) (← links)
- Mis-trafficking of bicarbonate transporters: implications to human diseases (Q37860156) (← links)
- Na(+) dependent acid-base transporters in the choroid plexus; insights from slc4 and slc9 gene deletion studies (Q38155158) (← links)
- Immunocytochemical localization of the translocase of the outer mitochondrial membrane (Tom20) in the human cochlea (Q38399634) (← links)
- Functional assessment of SLC4A11, an integral membrane protein mutated in corneal dystrophies (Q38734404) (← links)
- Multifunctional ion transport properties of human SLC4A11: comparison of the SLC4A11-B and SLC4A11-C variants (Q38748793) (← links)
- Corneal dystrophy-causing SLC4A11 mutants: suitability for folding-correction therapy (Q38986089) (← links)
- Vps35-deficiency impairs SLC4A11 trafficking and promotes corneal dystrophy (Q41866630) (← links)
- Dietary supplementation of boron differentially alters expression of borate transporter (NaBCl) mRNA by jejunum and kidney of growing pigs (Q42481804) (← links)
- Mouse Slc4a11 expressed in Xenopus oocytes is an ideally selective H+/OH- conductance pathway that is stimulated by rises in intracellular and extracellular pH. (Q46370015) (← links)
- Expression of SLC4A11 protein in mouse and rat medulla: a candidate transporter involved in outer medullary ammonia recycling (Q64280967) (← links)
- Slc4a11 disruption causes duct cell loss and impairs NaCl reabsorption in female mouse submandibular glands (Q91946288) (← links)
- Harboyan syndrome: novel SLC4A11 mutation, clinical manifestations, and outcome of corneal transplantation (Q98945076) (← links)
- Boron improves cardiac contractility and fibrotic remodeling following myocardial infarction injury (Q100533768) (← links)
- Borax induces osteogenesis by stimulating NaBC1 transporter via activation of BMP pathway (Q103028835) (← links)